Titlebook: Glioma Signaling; Jolanta Barańska Book 2020Latest edition Springer Nature Switzerland AG 2020 secondary messengers.nucleotide receptors.t

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Signaling Determinants of Glioma Cell Invasion,t therapeutic modalities, including anti-angiogenic therapy and radiotherapy, can enhance glioma invasiveness. Glioma cell invasion is stimulated by both autocrine and paracrine factors that act on a large array of cell surface-bound receptors. Key signaling elements that mediate receptor-initiated

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Receptor Tyrosine Kinases: Principles and Functions in Glioma Invasion, (RTK) is a tyrosine kinase located at the cellular membrane and is activated by binding of a ligand via its extracellular domain. Protein phosphorylation by kinases is an important mechanism for communicating signals within a cell and regulating cellular activity; furthermore, this mechanism functi

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STAT Signaling in Glioma Cells,ne and growth factor receptor signaling. The canonical JAK/STAT signaling pathway involves the activation of Janus kinases (JAK) or growth factors receptor kinases, phosphorylation of STAT proteins, their dimerization and translocation into the nucleus where STATs act as transcription factors with p

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Cannabinoid Signaling in Glioma Cells,nces and endogenous cannabinoids, such as anandamide and 2-arachidonoylglycerol. Cannabinoids elicit a wide range of central and peripheral effects mostly mediated through cannabinoid receptors. There are two types of specific G.-protein-coupled receptors cloned so far, called CB1 and CB2, although