Titlebook: Genetically Defined Animal Models of Neurobehavioral Dysfunctions; Peter Driscoll Book 1992 Springer Science+Business Media New York 1992

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https://doi.org/10.1007/978-3-322-92431-5epsy and antiepileptic drugs has mostly been done in mice and rats, in which seizures were induced by chemical or electrical means (Purpura et al., 1972; Löscher and Schmidt, 1988). Although these rodent models have proved useful for the development of new antiepileptic drugs, they are obviously not

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Die Grenzen der Logik sind erst der Anfang,o use a drug because of environmental influences or societal attitudes, but even when drugs are readily available and the societal attitudes permissive or supportive not everyone will use a drug and not everyone who experiments with the drug becomes dependent. Whenever individual differences of this

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https://doi.org/10.1007/978-3-8348-8205-9 in a report from the National Academy of Sciences reviewing the present state of our knowledge with regard to biological and behavioral aspects of aggression and violence, and recommending directions for future research in these areas. Research on genetic variants for different types of aggression

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https://doi.org/10.1007/978-3-658-23637-3 environmental conditions. The “coping” processes involve complex interactions among cognitive mechanisms, central neurochemical changes, and neuroendocrine responses, all processes aimed at improving readily available energy and at triggering adequate behavioral responses. Considerable variations a

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Genetically Defined Myelin Disorders,s of axon called nodes. Each myelinated segment is known as an internode. Myelin occurs in vertebrate nervous systems in two main forms, central and peripheral. It also occurs in some invertebrates in a form that resembles the myelin found in the peripheral nervous system (PNS) of vertebrates (Raine, 1984a).