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Titlebook: Gene Therapy of Cancer; Peter Walden,Uwe Trefzer,Patricia Zambon Book 1998 The Editor(s) (if applicable) and The Author(s), under exclusiv

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Satellite Data and Climate Models tumorigenicity of murine and human malignant cells by both p53 dependent and independent pathways (1–3, for a review see reference 4). Given the fact that the E1a protein is expressed at high levels during adenovirus 5 infection and due to its highly cytotoxic potential 5, we decided to study the e
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https://doi.org/10.1007/978-4-431-53985-8[1]. ErbB-2 phosphorylates and interacts with Shc that participates inbetween active tyrosine protein kinases to Ras signaling pathway. A point mutation or an elevated expression of ErbB-2 results in oncogenic growth of cells.
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Economic rationality in climate governance,ino derivatives; the latter is a potent cytotoxic agent, which following further non-enzymatic reaction with thioesters, leads to the generation of interstrand crosslinks in DNA [1–4] (Figure 1a). These cannot be efficiently repaired by the cell, and lead to cell death. Nitroreductase and CB1954 are
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978-1-4613-7444-2The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
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Gene Therapy of Cancer978-1-4615-5357-1Series ISSN 0065-2598 Series E-ISSN 2214-8019
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