找回密码
 To register

QQ登录

只需一步,快速开始

扫一扫,访问微社区

Titlebook: G Protein-Coupled Receptor Kinases; Vsevolod V. Gurevich,Eugenia V. Gurevich,John J.G. Book 2016 Springer Science+Business Media New York

[复制链接]
查看: 19383|回复: 55
发表于 2025-3-21 17:11:45 | 显示全部楼层 |阅读模式
书目名称G Protein-Coupled Receptor Kinases
编辑Vsevolod V. Gurevich,Eugenia V. Gurevich,John J.G.
视频video
概述Features detailed examinations of known GRK-dependent mechanisms, both associated with GPCR functions and receptor-independent.Covers a wide range of studies from invertebrates to humans.Provides prac
丛书名称Methods in Pharmacology and Toxicology
图书封面Titlebook: G Protein-Coupled Receptor Kinases;  Vsevolod V. Gurevich,Eugenia V. Gurevich,John J.G. Book 2016 Springer Science+Business Media New York
描述This collection explores up-to-date descriptions of known G protein-coupled receptor kinase (GRK)-dependent mechanisms, both associated with G protein-coupled receptor (GPCR) functions and the receptor-independent. The chapters cover a wide range of studies from invertebrates to humans, with sections of the volume covering GRK structure, mechanisms of activation, and interaction with GPCRs, GRKs in cell signaling, as well as physiological and pathophysiological mechanisms regulated by GRKs.  Written for the .Methods in Pharmacology and Toxicology. series, this book features the kind of practical detail necessary for success in the laboratory. Authoritative and timely, .G Protein-Coupled Receptor Kinases. features the kind of comprehensive mechanistic elucidation of GRK functions and their regulation in cells necessary for a better understanding of cell biology as well as for devising novel research approaches and therapeutic strategies..
出版日期Book 2016
关键词G protein-coupled receptors (GPCRs); Cell signaling; Pathophysiological regulation; GPCR signaling; GRK
版次1
doihttps://doi.org/10.1007/978-1-4939-3798-1
isbn_softcover978-1-4939-8142-7
isbn_ebook978-1-4939-3798-1Series ISSN 1557-2153 Series E-ISSN 1940-6053
issn_series 1557-2153
copyrightSpringer Science+Business Media New York 2016
The information of publication is updating

书目名称G Protein-Coupled Receptor Kinases影响因子(影响力)




书目名称G Protein-Coupled Receptor Kinases影响因子(影响力)学科排名




书目名称G Protein-Coupled Receptor Kinases网络公开度




书目名称G Protein-Coupled Receptor Kinases网络公开度学科排名




书目名称G Protein-Coupled Receptor Kinases被引频次




书目名称G Protein-Coupled Receptor Kinases被引频次学科排名




书目名称G Protein-Coupled Receptor Kinases年度引用




书目名称G Protein-Coupled Receptor Kinases年度引用学科排名




书目名称G Protein-Coupled Receptor Kinases读者反馈




书目名称G Protein-Coupled Receptor Kinases读者反馈学科排名




单选投票, 共有 1 人参与投票
 

0票 0.00%

Perfect with Aesthetics

 

0票 0.00%

Better Implies Difficulty

 

0票 0.00%

Good and Satisfactory

 

0票 0.00%

Adverse Performance

 

1票 100.00%

Disdainful Garbage

您所在的用户组没有投票权限
发表于 2025-3-21 22:17:54 | 显示全部楼层
发表于 2025-3-22 00:30:41 | 显示全部楼层
Rainer-Peter Meyer,Fabrizio Moro,Fritz Heftihis chapter summarizes the current state of knowledge, discusses the relationship between GRK1 and GRK7 in the context of . disease, and pinpoints fruitful future directions for advancement of the vision research field.
发表于 2025-3-22 05:40:18 | 显示全部楼层
发表于 2025-3-22 11:14:09 | 显示全部楼层
发表于 2025-3-22 15:03:08 | 显示全部楼层
https://doi.org/10.1007/3-540-33316-9 cAMP signaling to Epac/PKCε/ERK- mediated pathway. Via mechanisms that remain to be elucidated, low monocyte GRK2 leads to IL-10 deficiency and prolonged inflammatory pain. The clinical relevance of these findings is discussed in the light of the observed decrease of GRK2 in nociceptors, glia and l
发表于 2025-3-22 18:55:44 | 显示全部楼层
发表于 2025-3-23 00:53:58 | 显示全部楼层
发表于 2025-3-23 05:20:06 | 显示全部楼层
100 Keywords Wirtschaftsprüfung, contractility, and blood pressure, in response to catecholamines and other neurohormones. For these reasons, GPCRs are dynamically regulated to prevent overstimulation that could lead to cardiac diseases like heart failure (HF). This dampening process, known as desensitization, is initiated throug
发表于 2025-3-23 07:27:24 | 显示全部楼层
 关于派博传思  派博传思旗下网站  友情链接
派博传思介绍 公司地理位置 论文服务流程 影响因子官网 SITEMAP 大讲堂 北京大学 Oxford Uni. Harvard Uni.
发展历史沿革 期刊点评 投稿经验总结 SCIENCEGARD IMPACTFACTOR 派博系数 清华大学 Yale Uni. Stanford Uni.
|Archiver|手机版|小黑屋| 派博传思国际 ( 京公网安备110108008328) GMT+8, 2025-5-26 01:53
Copyright © 2001-2015 派博传思   京公网安备110108008328 版权所有 All rights reserved
快速回复 返回顶部 返回列表