Titlebook: Erythropoietin; Wolfgang Jelkmann,Andreas J. Gross Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 anemia.blood.cells.c

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Detoxification

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Erythropoietin, Blood Viscosity, and Hypertension: Implications for Patients with End-Stage Renal Dicorrecting this anaemia in patients maintained by haemodialysis [1–6]. Up to now, patients have been treated for more than 2 years with sustained benefit and without any evidence of loss of efficacy. Nevertheless, the development or aggravation of hypertension in a certain number of patients treated

Mortar

Recombinant Human Erythropoietin and ,-Retinoic Acid Therapy of Anemia in Hemodialysis and Peritonea the deficient production of erythropoietin (EPO) by the kidney [1–3]. This is confirmed by the very recent results concerning recombinant human erythropoietin (rHuEPO) use in uremic dialysis patients, which showed that intravenous administration of this hormone leads to a significant improvement in

Entrancing

别炫耀

mutineer

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Physiological Studies of Erythropoietin in Plasmad production of the hormone which in turn stimulates the bone marrow to increase the output of new red cells. Thus oxygen delivery is restored to normal. Hyperoxia leads to an increased oxygen supply to the sensor with decreased production of EPO (Fig. 1).

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HATCH

Erythrocytosis in Renal Graft Recipientsovercome their anemic state; their hematocrits once again begin to build up to become normal or close to normal [6, 7]. However, in some patients the hematocrit continues to rise even after complete correction of anemia, resulting in post-transplant erythrocytosis (PTE) [6–9].