Titlebook: Early Life Origins of Health and Disease; E. Marelyn Wintour,Julie A. Owens Book 2006 The Editor(s) (if applicable) and The Author(s), und

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https://doi.org/10.1007/978-3-658-31992-2etal programming recent experimental studies suggest that leptin and insulin resistance are critical endocrine defects in the pathogenesis of programming-induced obesity and metabolic disorders. Recent work has also shown that offspring of undernourished mothers display reduced locomotor activity in

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https://doi.org/10.1007/978-3-662-29437-6tions between small size at birth, neonatal catch-up growth and later metabolic disease. In this chapter we will review what has been learnt from human and experimental studies about the mechanistic basis for poor metabolic homeostasis following restricted fetal growth and neonatal catch-up growth,

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https://doi.org/10.1007/978-3-663-05478-8vels amongst women of reproductive age, and indeed, in pregnant women, places in sharp focus our scant understanding of the ramifications of this on offspring health, not only in the immediate neonatal period but, as a result of the recent spotlight on the early origins of adult disease and syndrome

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