Titlebook: Early Life Origins of Health and Disease; E. Marelyn Wintour,Julie A. Owens Book 2006 The Editor(s) (if applicable) and The Author(s), und

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978-1-4419-3950-0The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines

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https://doi.org/10.1007/978-3-322-84357-9 Metabolic Syndrome. The fetal origins of adult disease hypothesis proposes that these associations reflect permanent changes in metabolism, body composition and tissue structure caused by undernutrition during critical periods of early development. An alternative hypothesis is that both small size

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https://doi.org/10.1007/978-3-662-13473-3an motor system. Even less is known about how an adverse intrauterine event or environment might program motor learning, memory and function throughout the lifespan. We are only beginning to investigate how events during development of the brain and central nervous system might predispose some indiv

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https://doi.org/10.1007/978-3-7091-5363-5ulthood. Epigenetic mechanisms, which regulate the broad diversity of tissue-specific gene expression, are established during development and largely maintained throughout adulthood. Hence, to the extent that nutrition during development affects the establishment of epigenetic gene regulatory mechan

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