Titlebook: Early Indicators Early Treatments Neuroprotection in Multiple Sclerosis; Otto R. Hommes,Giancarlo Comi Book 2004 Springer-Verlag Italia 20

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Autoimmune Inflammation and Multiple Sclerosis,edness was the natural result of blood vessel physiology. Now we know that the blood vessels are only one of the many factors involved in inflammation, a process in which immune cells of various types and their molecular products interact with signal molecules produced by the injured tissue.

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Early Treatment of Progression in Multiple Sclerosis,tting (RR) to the progressive stages. It is becoming increasingly clear that progressive multiple sclerosis (MS) is associated with axonal loss and that axonal damage occurs early during RRMS. Disease progression thus develops well in advance of clinical progression and remains subclinical because,

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Imaging for Tissue Characterization in Multiple Sclerosis and Other White Matter Diseases,time. However, conventional MRI is not able to characterize and quantify the tissue damage within and outside these lesions. Other quantitative MR techniques have the potential to overcome such limitation. Among these techniques, MR spectroscopy (MRS), magnetization transfer imaging (MTI), and diffu

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Early Treatment in Multiple Sclerosis with Intravenous Immunoglobulin: Rationale and Study Design,udies of the natural course of the disease showed that disability will accumulate in more that 90% of patients and additional relapses may result in significant handicap [2]. Assessment of patients with a diagnosis of probable MS with positive brain magnetic resonance imaging (MRI) demonstrated that

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T1-Hypointense Lesions (T1 Black Holes) in Mild-to-Moderate Disability Relapsing Multiple Sclerosishe more non-specific T2-hyperintense lesions, which show greater signal intensity than normal brain on T2-weighted magnetic resonance imaging (MRI). The T1-hypointense lesions are areas of axonal loss, as well as matrix disruption [1, 2]. T1-hypointense lesions are moderately correlated with focal r

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