Titlebook: Early Indicators Early Treatments Neuroprotection in Multiple Sclerosis; Otto R. Hommes,Giancarlo Comi Book 2004 Springer-Verlag Italia 20

期满

brain damage accumulates very early in the course of multiple sclerosis. This book reviews the main neurobiological, magnetic resonance imaging, and clinical aspects of the early phases of the dis­ ease. Mechanisms ofirreversible axonal damage and the role played by the inter­ action of glia and the

Foreknowledge

https://doi.org/10.1007/978-3-540-37639-2is might be explained by observations, especially from magnetic resonance imaging (MRI) studies, that the correlation of inflammation with disease progression and development of disability is poor [1].

Instantaneous

at axonal damage occurs early during RRMS. Disease progression thus develops well in advance of clinical progression and remains subclinical because, due to compensatory mechanisms, impairment does not interfere with daily living activities at that stage.

Harrowing

Gobble

these three MR techniques to the understanding of the evolution of WMD, with a special focus on multiple sclerosis (MS). The application of MR techniques to the study of MS has indeed dramatically changed our understanding of how MS causes irreversible deficits and can serve as a useful model to be applied to other WMD.

left-ventricle

nflammation. Some forms of cell death in the ischemic penumbra also involve apoptosis [1, 2]. Studies in patients using positron emission tomography, combined perfusion and diffusion magnetic resonance imaging (MRI), and MR spectroscopy indicate that the penumbra in humans may remain viable for several (up to 48) hours after stroke onset [3-6].

发源

Imaging for Tissue Characterization in Multiple Sclerosis and Other White Matter Diseases,these three MR techniques to the understanding of the evolution of WMD, with a special focus on multiple sclerosis (MS). The application of MR techniques to the study of MS has indeed dramatically changed our understanding of how MS causes irreversible deficits and can serve as a useful model to be applied to other WMD.

FILTH

Neuroprotection in Acute Ischemic Stroke: Lessons for Early Treatment in Multiple Sclerosis,nflammation. Some forms of cell death in the ischemic penumbra also involve apoptosis [1, 2]. Studies in patients using positron emission tomography, combined perfusion and diffusion magnetic resonance imaging (MRI), and MR spectroscopy indicate that the penumbra in humans may remain viable for several (up to 48) hours after stroke onset [3-6].

Surgeon

Neuroprotective Treatment in Primary Progressive Multiple Sclerosis: a Phase I/II Study with Riluzois might be explained by observations, especially from magnetic resonance imaging (MRI) studies, that the correlation of inflammation with disease progression and development of disability is poor [1].

水獭

Early Treatment of Progression in Multiple Sclerosis,at axonal damage occurs early during RRMS. Disease progression thus develops well in advance of clinical progression and remains subclinical because, due to compensatory mechanisms, impairment does not interfere with daily living activities at that stage.