Titlebook: Dietary Fat and Cancer; Genetic and Molecula American Institute for Cancer Research Book 1997 The Editor(s) (if applicable) and The Author(

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,The Role of Peroxisome Proliferator Activated Receptor α in Peroxisome Proliferation, Physiologicalaminobenzidine and historically accounts for the name “peroxisomes”.. Peroxisome proliferation can result in an excess of H.O. that can potentially result in toxicity as discussed below. Under usual circumstances H.O. is decomposed to molecular oxygen and water by catalase and glutathione peroxidase

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A Hypothetical Mechanism for Fat-Induced Rodent Hepatocarcinogenesis,tabolism including their breakdown,. storage. and synthesis.. The complexity of the PPAR activation pathway has been substantially enhanced by the demonstration that PPAR DNA binding is linked to heterodimerization with a member of the retinoid X (RXR) family of receptors,. and more recent PPAR tran

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Short Chain Fatty Acid Regulation of Intestinal Gene Expression, of hematopoietic origin. We will also emphasize our own studies using the HT-29 colon adenocarcinoma cell line as a model for study of early cellular and molecular events associated with butyrate-mediated growth and differentiation.

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Regulation of Peroxisomal Fatty Acyl-CoA Oxidase in the Yeast,hese include enzymes of the peroxisomal ß-oxidation cycle. This regulation occurs at the transcription level, and is controlled by specific .-acting factors. The research in our laboratory has focused on the mechanisms involved in this regulation, and on the identification and characterization of th

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Session V,ily history of breast cancer alone (relative risk = 2.1) or than women with no family history of breast or ovarian cancer (relative risk = 1.1). Although there were few patients with a family history of both breast and ovarian cancer, these results supported a strong relationship between obesity, or

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Book 1997sures of dietary fat intake, variability in individual diets, and spe­ cies variations. Despite these limitations, there is a weak positive correlation between colon cancer and dietary fat intake, but with substantial differences for various ethnic groups. In the case of breast cancer, there is subs