Titlebook: Dietary Fat and Cancer; Genetic and Molecula American Institute for Cancer Research Book 1997 The Editor(s) (if applicable) and The Author(

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Short Chain Fatty Acid Regulation of Intestinal Gene Expression,t that analogous pathways have been defined for many other cell types, especially those in the hematopoietic lineages. Much of the published work on intestinal cells has focused on regulation by polypeptide growth factors and extracellular matrix proteins while relatively less attention has been giv

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Regulation of Gene Expression in Adipose Cells by Polyunsaturated Fatty Acids,orporated into the triacylglycerol droplet under lipogenic conditions while rapidly mobilizing them during lipolytic stimulation. Hence, the flux and magnitude of the fatty acid pool in adipocytes is constantly changing in response to hormonal, metabolic and genetic determinants. Due to the rapidly

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Regulation of Peroxisomal Fatty Acyl-CoA Oxidase in the Yeast,. Proliferation of this organelle, and induction of peroxisomal enzymes, is a phenomenon that occurs in diverse species, and is stimulated by a number of physiological and pharmacological stimuli. A large number of chemically diverse compounds, including hypolipidemic drugs and industrial plasticize

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Session V,raction of genetics and nutrition in cancer development. Sellers et al.. assessed the influence of body fat distribution and family history of breast and ovarian cancer on the risk of postmenopausal breast cancer. Linkage studies of the breast cancer susceptibility gene BRCA-1 on-chromosome 17p demo

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Abstracts,ncluding colon cancer. It is well established that fatty acid composition of a high fat diet is an important determining factor in exerting a tumor enhancing or an inhibitory effect. A variety of mechanism(s) has been proposed to explain how different lipids may modulate tumor growth. It is proposed

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Dietary Lipids and the Cancer Cascade,ssociated with large studies having sufficient power to detect an effect of diet on cancer incidence prohibit their implementation. In contrast to interventions with pharmaceutical or chemopreventive agents where intake of the drug is precisely controlled and compliance determined by an objective te

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Modulation of Intracellular Second Messengers by Dietary Fat during Colonic Tumor Development,o reduce the adverse effects of certain fats through several mechanisms, including dilution of carcinogens, decrease in transit time, reduction in fecal pH, chemical binding, and alteration in microflora metabolism.. However, experiments in animal models using a variety of fats and fibers have produ