Titlebook: Diastolic Relaxation of the Heart; Basic Research and C William Grossman (Herman Dana Professor of Medicin Book 1988 Martinus Nijhoff Publi

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O. -J. Grüsser,U. Grüsser-Cornehlsls. The sources of the calcium for this rise are the extracellular space and the sarcoplasmic reticulum. Within a specific range of concentration, in the vicinity of 1µ molar, the amount of force that is generated is dependent on the amplitude of the calcium concentration. The amplitude of the contr

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Fundamentals of Cell Physiology contractile force and is usually fully relaxed at this stage [1]. With continued impairment of adenosine triphosphate (ATP) synthesis, the ischemic myocardial cell begins to develop an increase in resting tension. This increase in resting tension could be due to a rise in cytosolic free Ca. ion con

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Alexander E. Stover,Philip H. Schwartzhen heart muscle contracts and relaxes isometrically, there is considerable internal shortening and lengthening during the rise and fall of tension development [2]. In-skeletal muscle, under those conditions where active muscle shortens and lengthens, the energetic cost of shortening is greater than

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Lyn Healy,Lesley Young,Glyn N. Staceyf ventricular filling pressure and volume. The advent of ultrasound and radionuclide techniques has rendered the assessment of diastolic function, i.e. filling rates and volumes, and systolic function still more common. While these technological developments have verified the notion of variable dias

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Justine D. Miller,Thorsten M. Schlaegertriphosphate (ATP) and creatine phosphate (CP) [2) reserves, an accumulation of protons [3], and a gain in Na. and loss of K. [4]. As the duration of the hypoxic episode progresses, other changes occur, including a gradual but sustained increase in end-diastolic resting tension. Many factors, includ

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https://doi.org/10.1007/978-3-642-16707-2me of onset, speed, and extent of the relaxation phase. Relaxation of the heart is defined by the events during systole by which the heart, as a muscle and as a pump, returns to a precontractile configuration. Relaxation of cardiac muscle includes isometric force decline and isotonic lengthening. Re

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Jeffrey K. McKee,Erica N. Chambersr muscle, these changes usually manifest themselves as alterations in force generation and in the rate of relaxation [2]. The studies described in this chapter were designed to answer the question of whether the mechanical abnormalities of hypertrophied muscle are related to changes in intracellular

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