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Titlebook: DNA Replication, Recombination, and Repair; Molecular Mechanisms Fumio Hanaoka,Kaoru Sugasawa Book 2016 Springer Japan KK 2016 DNA replicat

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发表于 2025-3-21 16:41:16 | 显示全部楼层 |阅读模式
书目名称DNA Replication, Recombination, and Repair
副标题Molecular Mechanisms
编辑Fumio Hanaoka,Kaoru Sugasawa
视频videohttp://file.papertrans.cn/261/260193/260193.mp4
概述Provides a comprehensive description of the 3Rs (DNA replication, recombination, and repair) network system.Highlights the relationship between molecular mechanisms and pathology.Explains the implicat
图书封面Titlebook: DNA Replication, Recombination, and Repair; Molecular Mechanisms Fumio Hanaoka,Kaoru Sugasawa Book 2016 Springer Japan KK 2016 DNA replicat
描述.This book is a comprehensive review of the detailed molecular mechanisms of and functional crosstalk among the replication, recombination, and repair of DNA (collectively called the "3Rs") and the related processes, with special consciousness of their biological and clinical consequences. The 3Rs are fundamental molecular mechanisms for organisms to maintain and sometimes intentionally alter genetic information. DNA replication, recombination, and repair, individually, have been important subjects of molecular biology since its emergence, but we have recently become aware that the 3Rs are actually much more intimately related to one another than we used to realize. Furthermore, the 3R research fields have been growing even more interdisciplinary, with better understanding of molecular mechanisms underlying other important processes, such as chromosome structures and functions, cell cycle and checkpoints, transcriptional and epigenetic regulation, and so on. This book comprises 7 parts and 21 chapters: Part 1 (Chapters 1–3), DNA Replication; Part 2 (Chapters 4–6), DNA Recombination; Part 3 (Chapters 7–9), DNA Repair; Part 4 (Chapters 10–13), Genome Instability and Mutagenesis; Part
出版日期Book 2016
关键词DNA replication; DNA recombination; DNA repair; Genome integrity; Chromosome; Epigenetic information; Mole
版次1
doihttps://doi.org/10.1007/978-4-431-55873-6
isbn_softcover978-4-431-56717-2
isbn_ebook978-4-431-55873-6
copyrightSpringer Japan KK 2016
The information of publication is updating

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The Fanconi Anemia Pathway and Interstrand Cross-Link Repairion, and the processes of ICL repair in the cell. In addition, we will highlight recent evidence that implicates endogenous aldehydes in creating genomic damage in FA cells, culminating in the FA phenotypes.
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https://doi.org/10.1007/978-1-4613-2261-0s stability dominates that of the overall genome and affects cellular functions, such as senescence. In this review, I will introduce the unique mechanisms by which the rDNA repetitive region and its physiological functions are maintained.
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Mismatch Repairritical intermediates. It has been mired by unseemly biochemical conditions and misinterpretation. The contemporary use of real-time single molecule imaging has the potential to finally and fully resolve the mechanics of MMR. This review describes genetic, biochemical, and biophysical studies that contributed to the development of models for MMR.
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Nonhomologous End-Joining accompanied by elucidation of the genetic basis of several types of severe combined immunodeficiency (SCID) and a better understanding of the mechanisms regulating the choice between HR and NHEJ. In this chapter, we will therefore discuss the NHEJ mechanism along with considerations on DSB repair pathway choice and associated disease phenotypes.
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