Titlebook: Current Topics in Heart Failure; Experimental and Cli Rainer W. Gülch,Gerolf Kissling Book 1991 Springer-Verlag Berlin Heidelberg 1991 expe

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书目名称Current Topics in Heart Failure影响因子(影响力)




书目名称Current Topics in Heart Failure影响因子(影响力)学科排名




书目名称Current Topics in Heart Failure网络公开度




书目名称Current Topics in Heart Failure网络公开度学科排名




书目名称Current Topics in Heart Failure被引频次




书目名称Current Topics in Heart Failure被引频次学科排名




书目名称Current Topics in Heart Failure年度引用




书目名称Current Topics in Heart Failure年度引用学科排名




书目名称Current Topics in Heart Failure读者反馈




书目名称Current Topics in Heart Failure读者反馈学科排名

遭受

Chemiluminescence as a marker of myocardial ischemiaburst and the actual scavenger state. Antioxidant application advantageously modifies the chemiluminescence effect, especially at the border zone. Induced chemiluminescence is a good method to characterize ischemically altered myocardial cellular metabolism.

窃喜

https://doi.org/10.1007/978-3-322-94268-5the IgM-isotype and fix complement in the acute stage of the disease. They also exhibit cytolytic and cytotoxic properties when incubated in vitro with isolated heart muscle cells. In addition, antigenic mimicry has been demonstrated to be operative, since they are cross-reactive to viral proteins.

Fantasy

Rentenrechnung und Äquivalenzprinzipime after infarction. Dilatation is compensatory first and normalizes stroke volume. However, left ventricular dilatation progresses without further hemodynamic profit and, thus, may participate in development of heart failure.

Immortal

GRIN

GRIN

鞭子

Experimental congestive heart failure due to myocardial infarction: Sarcolemmal receptors and cationive heart failure. Since depressed cardiac pump function is the hallmark of heart failure, it is suspected that decreased influx of Ca. into the cardiac cell is responsible for depressed contractile function. Because Ca. movements in the sarcolemmal membrane are known to involve Ca.-channels, Na.-Ca

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一加就喷出

Effects of nifedipine and moxonidine on cardiac structure in spontaneously hypertensive rats (SHR) —ent with nifedipine (27 mg/kg b.w./day) and the sympatholytic agent moxonidine (8 mg/kg b.w./day). The treated groups were compared with nontreated SHR and normotensive WKY (n =10 in each group)..When the therapy was started in 6-month old male SHR, blood pressure was increased and left ventricular