Titlebook: Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia; Proceedings of the 6 Alexander Baethmann,Nikolaus

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Disturbance of Endoplasmic Reticulum Functions: A Key Mechanism Underlying Cell Damage?,conclude that disturbance of the ER functions may be involved in stress-induced cell injury. In our view, ER calcium homeostasis plays an important role in maintaining the physiological state in cells balanced between the extremes of growth arrest and cell death on the one hand, and uncontrolled proliferation on the other.

AWE

https://doi.org/10.1007/978-3-540-89573-2conclude that disturbance of the ER functions may be involved in stress-induced cell injury. In our view, ER calcium homeostasis plays an important role in maintaining the physiological state in cells balanced between the extremes of growth arrest and cell death on the one hand, and uncontrolled proliferation on the other.

fatuity

Anemia

tolerance

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Medical Device Alarms - The Statistician,l LCMRglc at 2-h MCAo was near-normal, and its metabolism/flow ratio was elevated 4-fold above normal. By 1-h recirculation, however, LCMRglc throughout the prior zone of ischemia was depressed. Infarctive his-topathology was precisely determined by the antecedent LCBF decrement during ischemia: 70%

Scintigraphy

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Jiaxu Hong,Jianjiang Xu,Xinghuai Sun,Lei Cuiotected state, but has no adverse effects on hibernators and brain damage does not occur. In fact, even hippocampal slices from hibernating ground squirrels and cerebellar slices from anoxia-tolerant turtles show increased tolerance to a superimposed insult of aglycemia and hypoxia. Surprisingly, th

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奴才

https://doi.org/10.1007/978-3-642-14255-0particular discloses important indices of subsequent clinical evolution and outcome, thus adding to already well-known predictors such as age and severity of neurological status at hospital admission [20]. CT does have the aforementioned limitations regarding inter-observer agreement, which may, how