Titlebook: Computational Neuroscience of Drug Addiction; Boris Gutkin,Serge H. Ahmed Book 2012 Springer Science+Business Media, LLC 2012

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2197-1900 thora of theoretical schemes over the years to understand adDrug addiction remains one of the most important public health problems in western societiesand is a rising concern for developing nations. Over the past 3 decades, experimental research on theneurobiology and psychology of drug addiction h

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Performance of Missing Transverse Momentumthe empirical evidence for or against any particular model. These seven addiction models are then discussed in relation to the addictive phenomena for which they attempt to account and specific aspects of their feedback systems.

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L. Andrianopoli,R. D’Auria,M. Trigiantewell-known models under the effect of addictive drugs. We try to show how each of these partially complete models can explain some behavioral and neurobiological aspects of addiction, and why it is necessary to integrate these models in order to have a more complete computational account for addiction.

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ALIBI

Understanding Addiction as a Pathological State of Multiple Decision Making Processes: A Neurocomputwell-known models under the effect of addictive drugs. We try to show how each of these partially complete models can explain some behavioral and neurobiological aspects of addiction, and why it is necessary to integrate these models in order to have a more complete computational account for addiction.

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Aura231

Simple Deterministic Mathematical Model of Maintained Drug Self-administration Behavior and Its Phard to logical contradictions to other accepted facts. A quantitative pharmacological theory of self-administration behavior states that cocaine-induced lever pressing behavior occurs only when cocaine concentrations are within a certain range, termed the compulsion zone. These concentrations can be c

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Intermittent Adaptation: A Mathematical Model of Drug Tolerance, Dependence and Addictionis, which is demonstrated to fail in describing tolerance development to repeated drug administrations. The model assumes the development of tolerance to a repeatedly administered drug to be the result of a process of intermittently developing adaptation. The oral detection and analysis of endogenou

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