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Titlebook: Cognitive Enhancing Drugs; Jerry J. Buccafusco Book 2004 Birkhäuser Verlag 2004 Alzheimer.attention.cholinesterase.cns.cognitive disorders

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https://doi.org/10.1007/978-0-387-33244-4lcholinesterase (AChE), the enzyme that degrades synaptically-released acetylcholine. Despite this important application of the hypothesis, the origin of the cholinergic theory likely derives mainly from the effects produced by anticholinergic drugs, chiefly natural products like atropine and scopol
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https://doi.org/10.1007/978-0-387-33244-4gonists AMPA and NMDA, and the pharmacological intervention which should improve cognition in man. For details about receptor subtype classification and structure the reader is referred to recent reviews (e.g., [5]).
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Cognitive Enhancing Drugs978-3-0348-7867-8Series ISSN 2296-6056 Series E-ISSN 2296-6064
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https://doi.org/10.1007/978-0-387-33244-4ically developed countries the demand remains as strong as ever. This appeal is aided by the almost universal, though completely irrational assumption, that if a product is natural it must be safe. This chapter will consider those naturally-occurring substances that are believed to beneficially affect cognitive function.
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https://doi.org/10.1007/978-3-0348-7867-8Alzheimer; attention; cholinesterase; cns; cognitive disorders; functional brain imaging; learning and mem
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