Titlebook: Clinical Use of Calcium Channel Antagonist Drugs; Lionel H. Opie,William A. Coetzee Book 1989 Kluwer Academic Publishers, Boston 1989 angi

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Side Effects and Contraindications Drug Interactions and Combinations,re unless caused by hypertensive heart disease. Drug interactions of calcium antagonists occur with other cardiovascular agents such as α-adrenergic blockers, β-adrenergic blockers, digox in, quinidine, and disopyramide. The most marked interaction with digoxin is that with verapamil, which may rais

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Epilogue. Where Do We Go from Here?,tihypertensive properties established. Yet when one considers that the main action of all calcium antagonists is inhibition of vascular smooth muscle contraction, it is surprising that the antihypertensive effect has been so well disguised for so long. Probably this delay has been in part because th

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Book 1989n and com­ pare with the three first-liners? When the gloss is taken away from the advertisements, what is really left? The strong clinical bias of the present book should be complimented by further reading of books slanted towards fundamentals. One of the most important and recent of these is that

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https://doi.org/10.1007/978-3-319-02192-8to other ions and in their sensitivity to membrane potential and to drugs..It is not quite clear how the calcium current is changed during myocardial ischemia. Factors that may reduce the calcium current during ischemia are the increased extracellular potassium concentration, metabolic inhibition an

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https://doi.org/10.1007/978-3-319-02192-8ctivity, generally held to be a desirable property because the negative inotropic effect is usually a liability. The general clinical impression that nifedipine is the agent most active in vascular tissue in relation to the myocardial effect is supported by data on the relative potencies of these th

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