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Titlebook: Clinical Pharmacology of Learning and Memory; Walter B. Essman Book 1983 Spectrum Publications, Inc. 1983 pharmacology

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https://doi.org/10.1057/9781403977274ained from control rats of 10 months of age. The use of stabilized procaine hydrochloride (Gerovital H-2) in geriatric populations has been viewed as a possible route toward the use of this compound as an antidepressant or vehicle through which cognitive changes accompanying senility and arterioscle
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Mood Alterants,rmance of a nondiscriminative avoidance task by rats was potentiated by imipramine (10 mg/kg, i.p.) (Weissman, 1961). Several studies have indicated that the effects of imipramine in animals can be generally viewed as sedative (Battig, 1961; Hanson, 1961; Herr, et al., 1961; Maxwell and Palmer, 1961
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Ethanol, memory are to be ascribed to ethanol, then there are probably several forms of ethanol amnesia. Two forms have been described (Ryback, 1970) consisting of a state-dependent form and a “blackout” form.
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https://doi.org/10.1007/978-1-137-09193-2puts to the central nervous system alter the ionic equilibrium of the cell cytoplasm and modify the stability of the nitrogenous bases of the RNA molecule (Hydén, 1959). Altered molecular structure through base modification leads to the synthesis of new RNA-dependent proteins capable of interacting
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https://doi.org/10.3920/978-90-8686-752-3mine and in amnesic agents or events, but also from pharmacological studies. An early study (Woolley, 1965) found that mice with an elevated brain serotonin level produced with a combination of 5-hydroxytryptophan (60 mg/kg) and benzlmethoxytryptamine (15 mg/kg) had a 13% reduction in the average nu
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