Titlebook: Clinical Pharmacology of Learning and Memory; Walter B. Essman Book 1983 Spectrum Publications, Inc. 1983 pharmacology

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Procaine,rse depression, and promote learning and memory has warranted some brief consideration of its place in the clinical pharmacology of learning and memory. Learning and memory was assessed in rats chronically treated with procaine, using a 13-choice maze (Asian et al., 1965). Among control rats, 24-mon

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Papaverine and Hydergine,verine. This agent has been employed with varied success in the treatment of a variety of symptoms in the geriatric patient, including mental confusion (LeBrecque, 1966), chronic brain syndrome secondary to cerebral arteriosclerosis (Stern, 1970; Ritter et al., 1971; McQuillan et al., 1974), and psy

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Peptides,e retrieval of acquired responses. Several studies in animals have indicated facilitation of shuttle box avoidance in rats (Beatty et al., 1970) with ACTH using a high-intensity conditioning foot shock. Other studies (Stratton and Kastin, 1974) found that at a low level of foot shock in a shuttle bo

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https://doi.org/10.3920/978-90-8686-752-3mber of correct maze responses performed. A decrease in brain serotonin with either reserpine (1.2 mg/kg) or oral DL-phenylalanine combined with L-tyrosine increased correct maze responses by 7 and 9% respectively.

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Introduction: Mindscape and Landscape,e (estimated at approximately 1–2 .g/kg equivalents in the blood), given ten times over a 6-hour period, increased the onset of fatigue and did not affect vigilance or tracking behavior (Heimstra, 1962). Tracking performance was decreased by the inhalation of four cigarettes over a brief time period (Wenzel and Davis, 1961).