Titlebook: Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy; Santiago Grisolía,Vicente Felipo Book 1993 The Editor(s) (if applicable) and The Au

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Klaus Warm,Michael Metzler,H.-Joachim GlaserHepatic encephalopathy is one of the main causes of death in western countries. In Spain, nearly 12,000 people die every year by liver cirrhosis (-8,000) or other hepatic failures; this represents approximately 4% of the total number of deaths. In spite of much work, the actual cause of hepatic coma and death remains unclear.

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Control of Urea Synthesis and Ammonia Detoxification,Ammonia toxicity was first reported by Pavlov and coworkers a century ago (1). They excluded the liver from the circulation and found that when dogs treated in this way were fed meat, they developed hyperammonemia, which was associated with coma and led to the death of the animal.

morale

Activation of NMDA Receptor Mediates the Toxicity of Ammonia and the Effects of Ammonia on the MicrHepatic encephalopathy is one of the main causes of death in western countries. In Spain, nearly 12,000 people die every year by liver cirrhosis (-8,000) or other hepatic failures; this represents approximately 4% of the total number of deaths. In spite of much work, the actual cause of hepatic coma and death remains unclear.

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Dynamics of Rehabilitation Nursingate nitrogen by reduction of compounds containing nitrogen at its higher oxidation states to the -3 oxidation state (ammonia) which in turn is incorporated into amino acids in a reaction requiring removal of two electrons (1). Until relatively recently it was thought that in mammals all enzymatic re

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Robert S. Kern,Michael F. Greenension leads to one of the most serious dangers for the lives of these patients: upper digestive hemorrhage with its hemodynamic and metabolic sequels. The situation is dramatized by an ever — increasing circumstance: the ethylic origen of the hepatic lesion strikes in increasingly younger ages, or

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Johannes Lindenmeyer,Ulrike Dickenhorstd in many functions in the Central Nervous System. Thus, there is evidence that an increase in the effectiveness of glutamatergic synapses mediate some forms of synaptic plasticity which participate in processes of memory and learning. On the other hand, glutamate is involved in the brain damage ass