找回密码
 To register

QQ登录

只需一步,快速开始

扫一扫,访问微社区

Titlebook: Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy; Santiago Grisolía,Vicente Felipo Book 1993 The Editor(s) (if applicable) and The Au

[复制链接]
查看: 34731|回复: 48
发表于 2025-3-21 16:13:55 | 显示全部楼层 |阅读模式
书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy
编辑Santiago Grisolía,Vicente Felipo
视频video
丛书名称Advances in Experimental Medicine and Biology
图书封面Titlebook: Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy;  Santiago Grisolía,Vicente Felipo Book 1993 The Editor(s) (if applicable) and The Au
描述This volume contains the papers presented in the International Summer Course on "Cirrhosis, Hyperammonemia and Hepatic Encephalopathy," which was one of the prestigious Summer Course, of the Complutense University of Madrid held in EI Escorial, Spain, during August 10-14, 1992. Liver cirrhosis is one of the main causes of death in western countries. In addition there is a series of liver dysfunctions including fulminant hepatic failure, Reye‘s syndrome and congenital defects of urea cycle enzymes that could lead to hepatic encephalopathy, coma and death. As a consequence of impaired liver function, the ability to detoxify ammonia by its incorporation into urea is diminshed, resulting in increased ammonia levels in blood and brain. Hyperammonemia is considered one of the main factors in the mediation of hepatic encephalopathy and the classical clinical treatments are directed towards reducing blood ammonia levels. A part of the book is therefore devoted to the study of certain aspects of ammonia metabolism such as the regulation of the urea cycle, the main mechanism of ammonia detoxification in mammals, which is located mainly in the liver. The metabolism of ammonia in other tissues
出版日期Book 1993
关键词brain; death; liver; metabolism; nutrition; tissue; hepatology
版次1
doihttps://doi.org/10.1007/978-1-4615-2484-7
isbn_softcover978-1-4613-6058-2
isbn_ebook978-1-4615-2484-7Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy影响因子(影响力)




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy影响因子(影响力)学科排名




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy网络公开度




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy网络公开度学科排名




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy被引频次




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy被引频次学科排名




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy年度引用




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy年度引用学科排名




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy读者反馈




书目名称Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy读者反馈学科排名




单选投票, 共有 0 人参与投票
 

0票 0%

Perfect with Aesthetics

 

0票 0%

Better Implies Difficulty

 

0票 0%

Good and Satisfactory

 

0票 0%

Adverse Performance

 

0票 0%

Disdainful Garbage

您所在的用户组没有投票权限
发表于 2025-3-21 22:29:59 | 显示全部楼层
发表于 2025-3-22 00:56:18 | 显示全部楼层
发表于 2025-3-22 06:33:04 | 显示全部楼层
Two Different Families of NMDA Receptors in Mammalian Brain: Physiological Function and Role in Neuns either through activation of entry of extracellular calcium (Ca.) into the neuronal space or through the release of intracellular Ca. from internal stores (6–9). The events initiated by the activation of special subpopulations of glutamate receptors are thought to be instrumental in the long-term
发表于 2025-3-22 12:04:46 | 显示全部楼层
Ganglioside GM1 and its Semisynthetic Lysogangliosides Reduce Glutamate Neurotoxicity by a Novel Me metabotropic) which differ by the mechanism attending the transduction of the transmitter signals into specific receptor responses. Both transduction mechanisms generate a metabolic event of high functional significance for the receiving neuron. When metabotropic receptors (Fig. 1) are activated by
发表于 2025-3-22 15:42:09 | 显示全部楼层
Dynamics of Rehabilitation Nursingmammals can catalyze formation of NO (nitric oxide, +2 oxidation state) from the co-nitrogen (i.e. =NH.) of L-arginine in a fascinating reaction that requires a five electron transfer. The free radical NO is involved in the physiological functioning of the mammalian cardiovascular system, immune sys
发表于 2025-3-22 17:11:12 | 显示全部楼层
Johannes Lindenmeyer,Ulrike Dickenhorst in the effects of synaptically released GABA or glycine on neurons, and without changes in excitatory synaptic transmission. The inactivation of Cl-extrusion and resulting dysfunction of gate-inhibition is sufficient to produce in experimental animals changes of the EEG. The brain tissue levels of
发表于 2025-3-22 22:04:14 | 显示全部楼层
发表于 2025-3-23 03:08:13 | 显示全部楼层
Rehabilitation After Limb Salvage Surgery metabotropic) which differ by the mechanism attending the transduction of the transmitter signals into specific receptor responses. Both transduction mechanisms generate a metabolic event of high functional significance for the receiving neuron. When metabotropic receptors (Fig. 1) are activated by
发表于 2025-3-23 06:59:23 | 显示全部楼层
0065-2598 ion of the urea cycle, the main mechanism of ammonia detoxification in mammals, which is located mainly in the liver. The metabolism of ammonia in other tissues978-1-4613-6058-2978-1-4615-2484-7Series ISSN 0065-2598 Series E-ISSN 2214-8019
 关于派博传思  派博传思旗下网站  友情链接
派博传思介绍 公司地理位置 论文服务流程 影响因子官网 SITEMAP 大讲堂 北京大学 Oxford Uni. Harvard Uni.
发展历史沿革 期刊点评 投稿经验总结 SCIENCEGARD IMPACTFACTOR 派博系数 清华大学 Yale Uni. Stanford Uni.
|Archiver|手机版|小黑屋| 派博传思国际 ( 京公网安备110108008328) GMT+8, 2025-5-5 11:18
Copyright © 2001-2015 派博传思   京公网安备110108008328 版权所有 All rights reserved
快速回复 返回顶部 返回列表