Titlebook: Cholesterol Transport Systems and Their Relation to Atherosclerosis; Armin Steinmetz,Hans Kaffarnik,Jürgen Schneider Conference proceeding

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Conference proceedings 1989 E polymorphism in cholesterol homeostasis. The combination of general outline form and very specific aspects of cholesterol transport will interest those in other disciplines following developments in the field, as well as those directly involved in lipoprotein research.

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https://doi.org/10.1007/978-3-642-82074-8sma lipoproteins may also bind to the extracellular matrix of the arterial wall. Both processes are dependent on the concentration of lipoproteins in the intima, which is influenced by the plasma lipoprotein level, the permeability of the lining of endothelial cells, and blood pressure.

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https://doi.org/10.1007/978-3-642-75092-2CAT), an enzyme which catalyzes the esterification of free cholesterol, and, secondly, plasma lipid transfer protein(s) (LTPs), which are involved in the exchange and transfer of CE, triacylglycerols (TG), and phospholipids between the lipoproteins.

Project

1431-7567 sk factors for the development of atherosclerosis and coronary heart disease. This volume examines the state of the art of lipoprotein subclass metabolism and its relation to these diseases. The authors also report on new developments concerning the role of lipoprotein recptors, macrophages and apol

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,Gestaltänderung technischer Gebilde,e. These polymorphisms can be used as neutral markers to genotype individuals and to follow the inheritance of the LDL receptor gene in families. About 80% of individuals are heterozygous for at least one of these RFLPs, and they are therefore potentially informative for DNA diagnosis.