Titlebook: Cerebral Ischemia; Molecular and Cellul Wolfgang Walz Book 1999 Springer Science+Business Media New York 1999 brain.brain injury.cell.cereb

苦涩

Spreading Depression Waves as Mediators of Secondary Injury and of Protective Mechanismsdamaged core areas into the surrounding penumbra in focal ischemia. Recently, it has also been shown that SD waves in healthy tissue can have a neuroprotective effect. For these reasons, there is renewed interest in SD and it’s propagation mechanisms.

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energy-consuming processes is mainly caused by the active transport of ions, which is necessary to compensate for the transmembrane ion currents that are part ofthe complex signaling processes in the brain. Ninety-five percent ofthe brain‘s ATP is derived from mitochondrial oxidative phosphorylatio

极深

Springer Series in Optical Sciencesstemic hypotension. Focal cerebral ischemia affects restricted brain regions and occurs in a wide variety of clinical settings but is most commonly a result of cerebral vascular atherosclerosis. Focal ischemia is more frequent than global ischemia.

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Mechanisms of Cerebral Ischemic Damagere a result of cerebral ischemia (Mohr et al., 1978). Global cerebral ischemia involves the entire brain and occurs during cardiac arrest or severe systemic hypotension. Focal cerebral ischemia affects restricted brain regions and occurs in a wide variety of clinical settings but is most commonly a

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Cell Swelling in Cerebral Ischemiaules surrounded by a semipermeable lipid membrane. This is speculated as being the prototypic cell—a spontaneously forming phospholipid bilayer membrane enclosing enough protein and nucleic acids to form a nonequilibrium, self-replicating entity (Tosteson, 1963b). Donnan forces arise because of the