Titlebook: Cerebral Ischemia; Molecular and Cellul Wolfgang Walz Book 1999 Springer Science+Business Media New York 1999 brain.brain injury.cell.cereb

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https://doi.org/10.1007/978-3-662-22270-6area, and are considered the resident macrophages of the brain (Kreutzberg, 1987; Lawson et al., 1990; Perry and Gordon, 1988). At present, there are no known microglial subtypes as in the case of astrocytes.

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Activated and Phagocytic Microgliaarea, and are considered the resident macrophages of the brain (Kreutzberg, 1987; Lawson et al., 1990; Perry and Gordon, 1988). At present, there are no known microglial subtypes as in the case of astrocytes.

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Book 1999s a coordinator of vital homeostatic reflexes, and complex body reactions to external challenges, depends critically on the rate of oxygen delivery and oxygen consumption. Oxygen delivery depends on two variables described in the Fick relation­ ship: volume flow rate ofblood and the arterial oxygen

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he brain as a coordinator of vital homeostatic reflexes, and complex body reactions to external challenges, depends critically on the rate of oxygen delivery and oxygen consumption. Oxygen delivery depends on two variables described in the Fick relation­ ship: volume flow rate ofblood and the arterial oxygen 978-1-4757-4735-5978-1-59259-479-5

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https://doi.org/10.1007/978-3-662-22251-5vement of the permeant anions down their concentration gradients into the cells. However, this membrane potential will be reduced (less negative) when the membrane is also permeable to the cations whose equilibrium potential is zero. Thus, this situation is not stable; there will always be movement

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A. Cuschieri,L. K. Nathanson,S. M. Shimiur through necrotic and/or apoptotic mechanisms (Bennett and Huxlin, 1996). This review addresses our present understanding of perturbations of Cat. homeostasis in ischemia, sources of the lethal Cat. influx, activation of Cat.a-dependent processes in the neurotoxic cascade and Cat.-based therapeuti

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A. Cuschieri,G. Buess,J. Périssatmonstrated that about 2–5% of the electron nitric oxide (NO) flow in isolated brain mitochondria produces superoxide (O..) and hydrogen (H.O.) (Boveris and Chance, 1973). These constantly produced oxygen radicals are scavenged respectively by superoxide dismutases (SODs), glutathione peroxidases (GS