Titlebook: Cerebral Hemorrhage; Liang-Fu Zhou,Xian-Cheng Chen,Yi-Cheng Lu Conference proceedings 2008 Springer-Verlag Vienna 2008 aneurysm.brain.brai

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Concomitant intracerebral infusion of tissue plasminogen activator and thrombin leads to brain injurmbin cause brain injury when infused concomitantly with tissue plasminogen activator (tPA), which is used clinically to facilitate evacuation of intracerebral hematomas. In this study, we examined the effects of intracerebral infusion of tPA and thrombin, individually and in combination..Rats were i

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Blood-brain barrier function in intracerebral hemorrhagemark of ICH-induced brain injury. Such disruption contributes to edema formation, the influx of leukocytes, and the entry of potentially neuroactive agents into the perihematomal brain, all of which may contribute to brain injury. A range of factors have been implicated in inducing BBB disruption, i

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https://doi.org/10.1007/978-3-662-09482-2aldehyde, and superoxide dismutase, indicating a potent oxidizing environment. BOX concentrations increased from 0.084 ± 0.01 in fresh blood to 22.24 ± 4.28 in hematoma at 72h, and were 11.22 ±1.90 in adjacent white matter (nmol/g). Similar chemical and analytical results are seen in ICH in vivo, in

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