Titlebook: Cerebral Hemorrhage; Liang-Fu Zhou,Xian-Cheng Chen,Yi-Cheng Lu Conference proceedings 2008 Springer-Verlag Vienna 2008 aneurysm.brain.brai

雪上轻舟飞过

The antioxidant effects of melatonin after intracerebral hemorrhage in ratsoring and brain water content in the right basal ganglia was without significant difference between any treatment regimens (15 or 150mg=kg of melatonin) or time points of drug administration (15 min or 3 h post-ICH). Therefore, melatonin reduced oxidative stress but did not change extent of brain ed

碎片

Induction of autophagy in rat hippocampus and cultured neurons by irong showed an accumulation of MDC in cultured neurons exposed to ferrous iron..These results indicate that autophagy is induced by iron in neurons and that iron-induced autophagy may contribute to brain injury after ICH.

FLAT

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Intracerebral hemorrhage injury mechanisms: glutamate neurotoxicity, thrombin, and Srcnd ICH and improved behavioral function following ICH. This data leads us to suggest our hypothesis, that ICH, possibly via thrombin activation of protease-activated receptors, activates Src that phosphorylates NMDA receptors and other proteins that mediate injury after ICH.

Oratory

Increase in brain thrombin activity after experimental intracerebral hemorrhagene caused significant neuronal death at days 1 and 3, and resulted in significant brain tissue loss at day 28. These results suggest that thrombin inhibition in the acute phase may reduce ICH-induced brain damage.

cunning

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diathermy

alabaster

Treatment of stroke and intracerebral hemorrhage with cellular and pharmacological restorative therahis endogenous restorative brain plasticity process to potentiate functional recovery. The logic of restorative therapy is to treat intact or marginally compromised tissue and not injured or dying tissue. Thus, these treatments can be made available for all neurological injury. Once demonstrated to