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Titlebook: Cerebral Energy Metabolism and Metabolic Encephalopathy; David W. McCandless Book 1985 Plenum Press, New York 1985 Nervous System.anatomy.

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Cerebral Energy Metabolism and Metabolic Encephalopathy
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Carbon Dioxide Narcosiso unlike other endogenous metabolic end products (except ammonia), carbon dioxide is present in multiple chemical forms in the body (dissolved CO., H.CO., HCO. .). Both the absolute concentrations and the relative proportions of these forms are of great significance to a wide variety of tissue functions.
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Encephalopathy Due to Mercaptans and Phenolsdes and insecticides. More than 180,000 employees are potentially exposed to these compounds in the United States (NIOSH, 1978). Mercaptans are noted for their obnoxious odor, and methyl and ethyl derivatives have been put in piped natural gas in very low concentrations (0.02 parts per billion) to serve as warning agents for gas leaks.
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Thiamine Deficiency and Cerebral Energy Metabolisms a coenzyme as well as its possible role in nerve conduction. Following a consideration of the effects of thiamine deficiency on enzyme systems and metabolism will be a discussion of two major clinical entities thought to be associated with thiamine deficiency.
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opment and application of a number of techniques such as the 2-deoxyglucose method and microchemical methods for measuring metabolites and regional cerebral blood flow. Data from the application of these techniques are just beginning to be collected, and the next few years promise to bring many new
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Thiamine Deficiencyine and increases in glycine were also seen in the brains of thiamine-deprived animals and/or of those treated with pyrithiamine (Gaitonde and Nixey, 1974; Gabler et al., 1974; Gaitonde and Fayein, 1975).
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