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Titlebook: Cerebral Energy Metabolism and Metabolic Encephalopathy; David W. McCandless Book 1985 Plenum Press, New York 1985 Nervous System.anatomy.

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Anamnese und klinische Untersuchungents who survive. It is apparent that a more enlightened treatment of stroke will only evolve when a greater understanding is attained of the pathophysiological events that occur during and after a stroke. A variety of experimentally induced models of ischemia are now being used to investigate the p
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Book 1985ch are uniqve from those of any other region. The high-energy phosphate compounds ATP and phos­ phocreatine, supplied by the oxidative metabolism of glucose, are necessary for normal cerebral functions such as the maintenance of membrane potentials, transmission of impulses, and synthetic processes.
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atures which are uniqve from those of any other region. The high-energy phosphate compounds ATP and phos­ phocreatine, supplied by the oxidative metabolism of glucose, are necessary for normal cerebral functions such as the maintenance of membrane potentials, transmission of impulses, and synthetic processes.978-1-4684-1211-6978-1-4684-1209-3
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Hypoglycemia and Cerebral Energy Metabolismtechniques, and the results from these studies have been helpful in defining the sequence of events in hypoglycemic-induced coma. The purpose of this review is to examine normal glucose metabolism, models of experimental hypoglycemia, and some of the neurochemical aspects of hypoglycemia that have r
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Ischemic Encephalopathyents who survive. It is apparent that a more enlightened treatment of stroke will only evolve when a greater understanding is attained of the pathophysiological events that occur during and after a stroke. A variety of experimentally induced models of ischemia are now being used to investigate the p
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