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Titlebook: Cardiovascular Disease; Molecular and Cellul Linda L. Gallo Book 1987 Springer Science+Business Media New York 1987 Lipid.atherosclerosis.c

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书目名称Cardiovascular Disease
副标题Molecular and Cellul
编辑Linda L. Gallo
视频video
丛书名称Gwumc Department of Biochemistry and Molecular Biology Annual Spring Symposia
图书封面Titlebook: Cardiovascular Disease; Molecular and Cellul Linda L. Gallo Book 1987 Springer Science+Business Media New York 1987 Lipid.atherosclerosis.c
描述The Sixth Annual International Spring Symposium on Health Sciences, held in Washington, D. C. , in May 1986, brought together over 650 scientists from 19 countries to review and update research on cardiovascular disease. In this volume, which contains 59 chapters, an internationally recognized group of authors con­ tribute up-to-date accounts of molecular and cellular processes occurring in the vessel wall in atherogenesis and describe approaches to the prevention and treatment of atherosclerosis. The volume is divided into six major sections. Two sections deal with current aspects of lipoprotein metabolism. In Part I, we are alerted to the impact on li­ poprotein metabolism of structural heterogeneity within the four broad lipoprotein classes. Attention then turns to the components that orchestrate lipoprotein metab­ olism. Apolipoprotein identities, processing, and functions are described, as are the roles of lipid transfer proteins in plasma lipoprotein remodeling. Hepatic lipase synthesis and secretion are described. In Part II, Nobel Laureates Michael S. Brown and Joseph L. Goldstein describe mutations in the LDL receptor that reveal the functions of its various domains and po
出版日期Book 1987
关键词Lipid; atherosclerosis; cardiovascular; heart disease; lipoprotein; metabolism; mutation; protein; proteins;
版次1
doihttps://doi.org/10.1007/978-1-4684-5296-9
isbn_softcover978-1-4684-5298-3
isbn_ebook978-1-4684-5296-9
copyrightSpringer Science+Business Media New York 1987
The information of publication is updating

书目名称Cardiovascular Disease影响因子(影响力)




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Analysis of the , Translation Product of mRNA Coding for Chick Intestine Apolipoprotein A-It have the same molecular weight but different isoelectric points. After . translation of chick intestine mRNA, analysis of the primary translation product of immunoprecipitated apo A-I, by two-dimensional gel electrophoresis, also reveals the presence of at least three isoforms. The origin of these
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Genomic Structure, Biosynthesis, and Processing of Preproapolipoprotein C-IIycerides. Patients with deficiency of apo C-II have marked elevations of plasma triglyceride-rich lipoproteins and are at increased risk of pancreatitis. Apolipoprotein C-II has been cloned, and the complete genomic structure elucidated. The apo C-II gene consists of four exons interrupted by three
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Relationships of Changes in Postheparin Hepatic and Lipoprotein Lipase Activity to HDL-Cholesterol Cy men aged 30–59 before and after randomization to control status (C.. = 41) or to a 1-year weight loss program by moderate dieting without exercise (D, . = 38) or exercise (running) without dieting (E, . = 44). After 6 weeks of weight stabilization at 1 year, both D and E had lost significant (. >
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The Low-Density Lipoprotein Receptorcules and carry them into cells by receptor-mediated endocytosis. The human LDL receptor is a single-chain transmembrane glycoprotein of 839 amino acids. It specifically binds lipoproteins that contain apo B-100 and the active form of apo E. The ligand-binding domain comprises the 292 amino-terminal
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