Titlebook: Cardiovascular Disease; Molecular and Cellul Linda L. Gallo Book 1987 Springer Science+Business Media New York 1987 Lipid.atherosclerosis.c

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The Role of Apolipoprotein E and the Low-Density Lipoprotein Receptor in Modulating the , Metabolismate the role of apo E and the low-density lipoprotein (LDL) receptor in modulating the catabolism of lipoproteins containing apo B-48 and apo B-100, the following . metabolic studies were performed. The residence times of apo E., apo E., and apo E. were determined in normal subjects with a homozygou

伪善

Uptake of LDL-Sized Particles Extracted from Human Aortic Lesions by Macrophages in Cultured gel filtration. This lipoprotein, believed to be derived from plasma LDL that has accumulated in the arterial intima, possesses apo B immunoreactivity and is of the same size as LDL. However, compared to LDL, it is more electronegative, its protein-to-lipid ratio is lower, and its hydrated density

箴言

Derivatization of Apo-Low-Density Lipoprotein Lysine Residues by Lipid Peroxidation Products during LDL receptor of fibroblasts but is rapidly degraded by macrophages via the “scavenger” receptor pathway. The present studies were done to determine if these changes in receptor recognition might result from derivatization of lysine residues of apo B by products formed during the peroxidation of LDL

GENRE

Free Apolipoproteins A-I and A-IV Present in Human Plasma Displace High-Density Lipoprotein on Cultuoprotein-deficient serum (LPDS) to the incubation medium interferes with the binding in a concentration-dependent manner such that at a concentration of 6 mg protein/ml of LPDS almost a complete inhibition of the specific [.I]HDL binding is observed. Cultures of ABAE cells exposed to [.I]-labeled LP

Minatory

cognizant

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Jonathan L. Black-Branch,Dieter Flecks. In individuals with type III hyperlipidemia, the allele frequency for the RFLP detected with Xbal is significantly different from the allele frequency in normolipidemic individuals or in those with other types of hyperlipidemia. Within the normolipidemic population, homozygotes for the rare allel

OVERT

mercenary

https://doi.org/10.1007/978-94-6265-463-1ycerides. Patients with deficiency of apo C-II have marked elevations of plasma triglyceride-rich lipoproteins and are at increased risk of pancreatitis. Apolipoprotein C-II has been cloned, and the complete genomic structure elucidated. The apo C-II gene consists of four exons interrupted by three

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