Titlebook: Cardiomyocytes in Health and Disease; Chandrasekharan C. Kartha Book 2021 The Editor(s) (if applicable) and The Author(s), under exclusive

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Energy Metabolism in Cardiomyocyte The exact interacting mechanisms are unclear. As cardiomyocytes become terminally differentiated, its mitochondrial oxidative capacity increases and fatty acid β-oxidation is the major source of energy. Energy metabolism drives the post-natal development of cardiomyocyte and its complex architectur

tenosynovitis

commodity

Cardiomyocyte Response to Ischemic Injurygulation of apoptosis and inflammation in the peri-infarct area dictate maintenance of functional capacity of CMs. An understanding of the cellular pathways of death and survival of CMs is requisite for developing appropriate strategies to salvage CMs and preserve cardiac function during AMI.

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Cardiomyocytes in Heart Failureon. Both gradual and acute cell death via apoptosis, necrosis, and autophagy are hallmarks of HF. Given that the pathophysiology of HF involves death or dysfunction of CMs, understanding their mechanisms is imperative for identifying strategies for the treatment of HF and reducing related complicati

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great gains in attempts to identify disease biomarkers and therapeutic targets for better management of patients with heart diseases. Possibilities to induce regeneration or proliferation of cardiomyocytes and thus repair and or regenerate the damaged heart are also on the horizon. 978-3-030-85538-3978-3-030-85536-9

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