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Titlebook: Cardiac Valvular Medicine; Nalini M. Rajamannan Book 2013 Springer-Verlag London 2013 Cardiac Surgery.Cardiology.Internal Medicine

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https://doi.org/10.1007/978-94-007-5986-2acement is the number one indication for the treatment of this disease process (Bonow et al. 1998). For years, this disease has been described as a passive phenomena during which serum calcium attaches to the valve surface and binds to the leaflet to form nodules. Over decades, as aortic stenosis pr
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https://doi.org/10.1007/978-94-007-5986-2n coincide (Otto et al. 1994). Observed cardiovascular event rates might therefore differ substantially despite equal AS severity in the presence of additional vascular disease, such as atherosclerosis or hypertension (Otto et al. 1999; Briand et al. 2005). Moreover, cardiac response to the same pre
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The affine Coxeter transformation,as well as the study of the origin and progression of its disease from different points of view: molecular, anatomical, functional (Aikawa and Otto 2012). Calcium early detection and quantification plays a significant role in the study of calcified aortic valve disease (CAVD), as it has shown relati
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Springer Monographs in Mathematicslso includes reduced arterial compliance as well as alteration of LV geometry and function. This particular nature of the disease underlines the need for a more comprehensive assessment of AS severity going beyond the simple measurement of the conventional parameters of stenosis severity (i.e. peak
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Springer Monographs in Mathematicspediment to left ventricular outflow and provide relief from heart failure symptomatology. The procedure has been applied successfully in congenital aortic valve stenosis (Fratz et al. 2008) and has been studied extensively in calcific degenerative aortic stenosis in adults (Safian et al. 1988a; McK
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Springer Monographs in Mathematicsformation (Fig. 13.1) (Elmariah and Mohler 2010; Goldbarg et al. 2007; Mohler et al. 2001). Significant evidence supports the notion that calcification within heart valves is a manifestation of generalized atherosclerosis (Allison et al. 2006; Yamamoto et al. 2003; Pohle et al. 2001) and that these
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