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Titlebook: Cardiac Energetics; Basic Mechanisms and R. Jacob,Hj. Just,Ch. Holubarsch Conference proceedings 1987 Springer-Verlag Berlin Heidelberg 198

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楼主: Tyler
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Normal form results for default logic,h subdivisions are often thermodynamically misleading. There is an activation term that relates to Ca . release and retrieval, a work term and a stress or load-dependent heat term. The work and load-dependent energy terms presumably have their origin in the actin-activated myosin ATPase. It can be s
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Deduction with supernormal defaults,xotonic contractile work, depending on the preload, afterload and beat rate in isolated superfused left guinea pig atria. The mitochondrial activity was measured by (1) continuous determination of the O. uptake rate, (2) the rate of .CO. production from labelled glucose or FFA and (3) separate measu
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Vector Delay Differential Equations,n electrode..Using a diffusion model we eliminated the effect of oxygen storage on the measured time course of oxygen uptake to determine the time constant of mitochondrial “off” kinetics. Two different approaches were followed. In Method 1, two steady-state levels were compared, whereas in Method 2
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https://doi.org/10.1007/978-1-4614-3455-9dard one-pulse NMR spectroscopy coupled with conventional biochemical analyses provides information about the average cytosolic concentrations of ATP, creatine phosphate (CrP), creatine (Cr) and H. in the heart. By combining these techniques, we tested the hypothesis that the velocity of the creatin
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https://doi.org/10.1007/978-3-319-19518-6vary considerably and differences are also found in their myosin composition. In this study a comparison was made between the rate of ATP hydrolysis estimated from the stable maintenance heat rate and the mechanical performance of fibres of type 1, 2 and 3..In the experiments, firstly the force-velo
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Orthonormal Series and Approximation,rations were realized on representative samples of normotensive rats and spontaneously hypertensive rats (SHR) in compensated stages, as well as in SHR in a state of congestive cardiac failure. Cardiac dynamic measurements were performed under Urethane anaesthesia and open chest conditions. Myosin i
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