Titlebook: Cancer Stem Cell Resistance to Targeted Therapy; Cristina Maccalli,Matilde Todaro,Soldano Ferrone Book 2019 Springer Nature Switzerland AG

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lipoatrophy

Rajkumar Buyya,Lalit Garg,Sanjay MisraCSCs are endowed with the capability of initiating tumor growth and becoming dormant at distant organ sites just waiting for optimal conditions amenable for metastatic outgrowth. This cancer subpopulation is inherently resistant to anticancer therapeutics, and its targeting could avoid metastatic di

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https://doi.org/10.1007/978-3-031-05528-7d by a period of clinical complete remission after which the tumor eventually relapses. This course of events suggests that a small number of tumor cells evaded the therapeutic schedule and give rise to the recurrence, sometimes even after many years. Multiple processes have been advocated to underl

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Industrial IoT Technologies and Protocolsrmacological strategies for the treatment of advanced stage disease are in fact largely ineffective mostly due to the emergence of drug resistance. The cancer stem cell (CSC) hypothesis suggests that treatment failure and tumor relapse may be explained by the existence of a subset of self-renewing c

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ovation

https://doi.org/10.1007/978-3-030-58996-7d therapies. Melanoma genetic and functional heterogeneity is the major cause of therapy failure, with different subpopulations that can be spared by treatments and which then support tumor regrowth. Many studies have suggested different approaches to isolate and characterize cancer stem cells (CSCs

morale

https://doi.org/10.1007/978-3-642-73596-7mization in order to provide benefit to a broader number of cancer patients. In addition, since cancer stem cells/cancer-initiating cells represent the component of tumor resistant to therapeutic interventions, targeting of these cells is mandatory to achieve complete eradication of tumors..This cha

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