Titlebook: Cancer Stem Cell Resistance to Targeted Therapy; Cristina Maccalli,Matilde Todaro,Soldano Ferrone Book 2019 Springer Nature Switzerland AG

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A Cancer Stem Cell Perspective on Minimal Residual Disease in Solid Malignancies, a very elegant framework for MRD. However, the cancer stem cell theory is still an intense field of study, and many challenges remain that each has its own impact on how MRD can be explained as a consequence of therapy-resistant cancer stem cells. This chapter will provide an overview of the develo

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Cancer Stem Cells in Lung Cancer: Roots of Drug Resistance and Targets for Novel Therapeutic Strateome and response to therapies. We will next review evidences supporting the proposed mechanisms of resistance of CSCs to chemotherapy and targeted therapies and in particular intrinsic CSCs’ properties such as enhanced activation of the DNA damage repair machinery and anti-apoptotic signaling, incre

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Book 2019tific experts. The critical revision of available results in terms of immunological profile of CSCs/CICs and the efficacy in targeting these cells by immunological approaches, results in a comprehensive and up to date recapitulation of the field and provides interesting suggestions on how to focus f

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Industrial IoT Technologies and Protocolsome and response to therapies. We will next review evidences supporting the proposed mechanisms of resistance of CSCs to chemotherapy and targeted therapies and in particular intrinsic CSCs’ properties such as enhanced activation of the DNA damage repair machinery and anti-apoptotic signaling, incre

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Cryptography Concepts: Confidentiality,activities and cancer stem cell transcription factors, it was hypothesized that a crosstalk may exist between YY1 and CSCs transcription factors. Proteomic analysis was performed for the expression of YY1, SOX2, OCT4, and BM1 and delineated the presence of four groups of cancers with different molec

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A. Schneider,G. Meinhardt,E.-M. De Villiersntiate into multiple cell lineages, and initiate tumor formation. CSCs possess a distinct immunological profile compared with the bulk of the tumor and have evolved complex strategies to suppress immune responses through multiple mechanisms, including the release of soluble factors and the overexpre