Titlebook: Cancer Metastasis, Molecular and Cellular Mechanisms and Clinical Intervention; Wen G. Jiang,Robert E. Mansel Book 2000 Springer Science+B

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Gastrointestinal Tumours: Cancer Metastasis, Molecular and Cellular Biology,

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Cancer Metastasis, Molecular and Cellular Mechanisms and Clinical Intervention

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Metastatic Cascade, Molecular/Cellular Events and New Therapeutic Opportunities, have to successfully complete. The understanding of the biology of the process has enabled development of certain therapeutic measures that may eventually lead to the design of new anti-metastasis therapies.

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The E-Cadherin/Catenin Complex in Invasion: The Role of Ectodomain Shedding,a cytoplasmic part. The cytoplasmic part is connected with the actin cytoskeleton via the catenins. ß-catenin appears to be a functional modulator of the complex, because its association is not restricted to E-cadherin only. (ß-catenin is also found in cytoplasmic (e.g. with the Adenomatous Polyposi

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Cancer and Cancer Metastasis-Related Genes,s that normally encode for proteins which control the cell cycle, DNA repair and transcriptional events, the result is deregulation of cellular proliferation and other intracellular pathways, ultimately leading to tumour development.

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Hepatocyte Growth Factor and Met in Tumour Invasion-metastasis: From Mechanisms to Cancer Preventiovities. The utilization of the HGF-Met system in cancer cells confers invasive and metastatic potentials. HGF potently enhances dissociation of cells, cell-matrix interaction, extracellular matrix breakdown, invasion, and angiogenesis, all critical events in the metastatic cascade. Tumour-stromal in

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