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Titlebook: Calcium, Neuronal Function and Transmitter Release; Proceedings of the S Rami Rahamimoff,Bernard Katz Conference proceedings 1986 Martinus

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https://doi.org/10.1007/978-3-658-19608-0corded in the postsynaptic cell comes from experiments performed on the neuromuscular synapse. This preparation which is most suitable for electrophysiological studies is unfortunately not adequate for biochemical experiments. Therefore, we are using synaptosomes from brain (intact or following osmo
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https://doi.org/10.1007/978-3-658-17570-2toreceptor, remain reduced for days after associatively conditioning the nudibranch mollusc .. Because intracellular calcium, Ca.. (as measured by differential absorption spectrophotometry of calcium complexed with the dye arsenazo III), undergoes prolonged elevation during the acquisition of the co
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Die Wohnungsgemeinnützigkeit in Österreichrnal medium via voltage-regulated cation-selective channels (reviews, 1, 2, 3). At the vertebrate neuromuscular junction this dependence is manifest in essentially the same way for release provoked by nerve impulses (i.e., end-plate potentials, EPPs) or release manifest as a raised frequency of mini
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Soziale Ungleichheit von Jugendlichen,nated subsequent work on this problem. We have modified their model by adding one additional parameter: the level of Ca in the resting nerve terminal. This single addition substantially alters the behavior of the model and makes it necessary to reinterpret many previous measurements. We have present
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https://doi.org/10.1007/978-3-642-91120-0 in the extracellular solution (Jenkinson, 1957; Dodge & Rahamimoff, 1967). When the calcium concentration in the bathing solution is clamped at a constant level by employing a suitable calcium buffer, the average endplate potential amplitude and quantal content are reduced relative to those measure
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Wilfried von Bredow,Thomas Jägerarization-transmitter secretion coupling to occur (29). Nerve impulse-evoked multi-quantal release of acetylcholine (ACh), giving rise to the end-plate potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and
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