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Titlebook: Calcium, Neuronal Function and Transmitter Release; Proceedings of the S Rami Rahamimoff,Bernard Katz Conference proceedings 1986 Martinus

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书目名称Calcium, Neuronal Function and Transmitter Release
副标题Proceedings of the S
编辑Rami Rahamimoff,Bernard Katz
视频video
丛书名称Topics in the Neurosciences
图书封面Titlebook: Calcium, Neuronal Function and Transmitter Release; Proceedings of the S Rami Rahamimoff,Bernard Katz Conference proceedings 1986 Martinus
描述This symposium entitled Calcium, Neuronal Function and Transmitter Re­ lease, was in the framework of the regional meeting of the International Union of Physiological Sciences, that took place in Jerusalem between August 26-31, 1984. The symposium dealt with the role of calcium ions in regulation of a large number of important processes in modern neurobiology, from molecular and cellular points of view. In this context, we consider heart and most cells as ‘honorary neurons‘. The meeting was comprised of lectures and quite intense discussions. We hope that the transcription of the discussions which follow the articles will give the reader a feeling of the intense, but pleasant atmosphere that per­ vaded during this symposium. It is our pleasure to thank Mrs. Miriam Silber, the assistant editor of this book, for her hard work in transcribing the discussions, retyping large portions of the book, and getting the approval of the authors. We express our thanks to Dr. Halina Meiri and Dr. Simona Ginsburg for their editorial assis­ tance. The unfailing help of Ms. Rachel Klein and Ms. Shoshana Wineberg is greatly appreciated. xxvii SECTION 1 CALCIUM CHANNELS, TRANSPORTERS & CALCIUM REGULAT
出版日期Conference proceedings 1986
关键词biology; brain; neurobiology; neurons; physiology; receptor
版次1
doihttps://doi.org/10.1007/978-1-4613-2307-5
isbn_softcover978-1-4612-9420-7
isbn_ebook978-1-4613-2307-5Series ISSN 0897-3946
issn_series 0897-3946
copyrightMartinus Nijhoff Publishing, Boston 1986
The information of publication is updating

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Modulation Of Calcium Channels in Cardiac Cells by Neurotransmitters channels. Adrenergic neurotransmitter effects on Ca currents, I. in cardiac cells have served as prime examples of modulation of voltagegated ion channels. Recently it has become possible to analyze molecular events Involved in Ca channel modulation. Ca channels are activated by changes in membrane
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Persistent Inactivation of Potassium Currents by Elevation of Intracellular Calciumtoreceptor, remain reduced for days after associatively conditioning the nudibranch mollusc .. Because intracellular calcium, Ca.. (as measured by differential absorption spectrophotometry of calcium complexed with the dye arsenazo III), undergoes prolonged elevation during the acquisition of the co
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Some Consequences of Intracellular Calcium Binding on Phasic Synaptic Transmitter Releasear junction. Katz & Miledi (1) showed that external calcium was necessary at the time of the first nerve impulse and suggested that calcium entered the terminal and, by binding to some receptor site at the presynaptic terminal, released transmitter. The “residual calcium” hypothesis for facilitation
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Calcium Cooperativity in Calcium Entry and Calcium Action, and its Implications with Regard to Facilrnal medium via voltage-regulated cation-selective channels (reviews, 1, 2, 3). At the vertebrate neuromuscular junction this dependence is manifest in essentially the same way for release provoked by nerve impulses (i.e., end-plate potentials, EPPs) or release manifest as a raised frequency of mini
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