Titlebook: Breast Cancer: Biological and Clinical Progress; Proceedings of the C L. Dogliotti,A. Sapino,G. Bussolati Conference proceedings 1992 Sprin

disciplined

bleach

CHOKE

Stricture

ACTIN

c-,B-2, a Tyrosine Kinase Growth Factor Receptor and its Role in Breast Cancerligand binding domain. The group is subdivided into four subgroups based on differences in predicted structure (1). The type I subgroup includes the epidermal growth factor receptor (EGFR), encoded by the . gene, . and ., which have two cysteine rich domains within the extracellular region and a sin

wangle

Mammary Morphogenesis and Oncogenesudies have now appeared describing the use of different transgenes and several different promotor systems to produce mammary tumors (.,.). The use of binary systems for tissue-specific promotion of oncogenes and the production of bigenic and trigenic animals through hybrid crosses promise to expand

样式

Interdependence of Hormones and Growth Factors in Lobulo-Alveolar Development of the Mammary Gland ae growth factors such as epidermal growth factor (EGF), transforming growth factor alpha (TGF-a), insulin-like growth factors (IGF-I and IGF-II), fibroblast growth factors (FGF) [1], and possibly platelet derived growth factor (PDGF) [2]. The other is the negative pathway involving agents such as me

不可磨灭

The Role of Estrogen Regulated Secreted Proteins for Growth Regulation of Human Breast Canceretiology of the disease as a promoter for the growth of initiated cells and as a promotor for growth of the established tumor cells. A prerequisite for estrogen stimulated cell proliferation is presence of estrogen receptors in the tumor cells, and about 60% of the primary breast tumors contain estr

肿块

The Prolactin-Inducible Protein / Gross Cystic Disease Fluid Protein (PIP/GCDFP-15): Genetic Analysiuman breast cancer cell line in response to lactogenic peptide hormones (human prolactin and human growth hormone) and androgen, and independently by Haagensen and co-workers (3) as a protein found in abundance in the fluid of gross cystic disease of the breast. Haagensen and colleagues (3, 4) have

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