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Titlebook: Brain Edema IX; Proceedings of the N Umeo Ito (Director and Neurosurgeon in Chief),Alex Conference proceedings 1994 Springer-Verlag/Wien 19

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期刊全称Brain Edema IX
期刊简称Proceedings of the N
影响因子2023Umeo Ito (Director and Neurosurgeon in Chief),Alex
视频video
学科分类Acta Neurochirurgica Supplement
图书封面Titlebook: Brain Edema IX; Proceedings of the N Umeo Ito (Director and Neurosurgeon in Chief),Alex Conference proceedings 1994 Springer-Verlag/Wien 19
影响因子The first international symposium on brain edema was held in Vienna/ Austria in 1965 followed by altogether eight meetings since. The most recent was organized in Y okohama by the Department of Neurosurgery of the Musashino Red Cross Hospital, Tokyo. The continuing interest of both, clinicians and experimental scientists alike may be attributable to the fact that brain edema is a common denominator of many cerebral disorders, which under acute conditions threatens life and weIl-being of afflicted patients. Although progress in understanding as weIl as treatment can be recognized since 1965 many problems remain, particularly concerning the control of brain edema under acute conditions, as in trauma or ischemia. A quantum leap was the distinction of the cytotoxic and vasogenic brain edema prototypes as advanced by Igor Klatzo, providing for transition from a morphological to functional understanding now. The recent brain edema meetings were certainly benefiting from developments of both, molecular neurobiology on the one hand side and functional brain imaging at an ever-increasing resolution on the other, such as magnetic resonance imaging orpositron emission tomography. The internat
Pindex Conference proceedings 1994
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Astrocyte Swelling in Liver Failure: Role of Glutamine and Benzodiazepines MSO. Agonists of the peripheral-type BZD receptor aggravated ammonia-induced swelling, whereas a peripheral BZD receptor blocker, PK 11195, diminished the extent of swelling. Our findings implicate glutamine and the peripheral-type benzodiazepine receptor in the pathogenesis of the edema associated with fulminant hepatic failure.
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The Role of K+ Influx on Glutamate Induced Astrocyte Swelling: Effect of Temperature K. uptake as well as the glutamate effects on swelling. These findings show the importance of Ca. in the mechanism of glutamate swelling and suggests that glutamate induces swelling through stimulation of K+ influx and that the diminished effect of glutamate swelling with decrease in temperature may be due to a decrease in K+ uptake
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Guide to Clinical Workstation Useal environment. Current evidence suggests glial swelling to occur together with glutamate uptake or in response to extracellular acidosis. Glial swelling, therefore, is rather the result of homeostatic mechanisms than an indication of glial demise.
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Image Reconstruction and Review MSO. Agonists of the peripheral-type BZD receptor aggravated ammonia-induced swelling, whereas a peripheral BZD receptor blocker, PK 11195, diminished the extent of swelling. Our findings implicate glutamine and the peripheral-type benzodiazepine receptor in the pathogenesis of the edema associated with fulminant hepatic failure.
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