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Titlebook: Boundaries between Promotion and Progression during Carcinogenesis; Oscar Sudilovsky,Henry C. Pitot,Lance A. Liotta Book 1991 Plenum Press

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发表于 2025-3-21 16:49:12 | 显示全部楼层 |阅读模式
期刊全称Boundaries between Promotion and Progression during Carcinogenesis
影响因子2023Oscar Sudilovsky,Henry C. Pitot,Lance A. Liotta
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学科分类Basic Life Sciences
图书封面Titlebook: Boundaries between Promotion and Progression during Carcinogenesis;  Oscar Sudilovsky,Henry C. Pitot,Lance A. Liotta Book 1991 Plenum Press
影响因子The pUrpOSE! of this conference was not to define the two areas that are being bound, which might be a well nigh impossible proposition. Rather, its focus was to concentrate on the mechanistic similarities between promotion and progression. Are the areas involved within the boundaries a continuum? Are these two simultaneous processes? Or are some of the affected cells in the stage of promotion when at the same time others have undergone irreversi­ ble changes tha. t position them in the stage of progression? Or are these two stages the same thing, but called by different names? To explore such concepts we assembled investigators with various back­ grounds and asked them to specifically address these and other questions about "The Boundaries", within the context of the session to which they con­ tributed. The conference lasted two and a half days, from Wednesday to Friday. There were at least four speakers per session with morning and after­ noon sessions each day, except on Friday when the meeting ended at noon. The first day, each speaker had 25 minutes to present a position, followed by five minutes of discussion. At the end of the session there were 40 or 50 minutes of exchange
Pindex Book 1991
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发表于 2025-3-21 20:51:29 | 显示全部楼层
Interval 1: Grandma Baré’s Diaryulated proliferation. However, the appearance of a tumor is not really an endpoint in this process, and the more general term, progression, refers to the continuous evolution in characters both before and after a tumor has formed.
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Introduction to Loop Transformationst both genetic and epigenetic mechanisms are important. Altered growth control and differentiation leading to a more embryonic phenotype appear to be critical consequences of the genetic and epigenetic changes.
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Reformulating Imperial Authority,ion, while Dr. Slaga expressed the opinion that both processes exist as a continuum. So I would like to start the discussion by asking Dr. Pitot how he would contrast his belief with those of Dr. Slaga.
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https://doi.org/10.1007/978-1-349-02565-7 appear to persist in the embryonic portion of the embryo up until 7.5 days.. These undetermined pluripotent stem cells have been established as permanent tissue culture cell lines either directly from the embryo (embryonic stem (ES) cells). or indirectly from teratocarcinoma tumors (embryonal carcinoma (EC) cells) (See Fig. 1).
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https://doi.org/10.1057/9780230514003and experimental rodent models. Attempts to develop a cohesive picture of how these factors participate in mammary tumorigenesis have been hampered, in part, by a lack of information on the specific genetic lesions that contribute to the initiation and/or evolution of tumor development.
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Transkulturalität – Translation – Transfery, tumors induced by such viruses in animals are all polyclonal. Such viruses have never been found in healthy animals, a statement that cannot be made for retroviruses without . genes or DNA tumor viruses, which are commonly found in animals outside the laboratory and only transform cells indirectly and inefficiently..
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