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Titlebook: Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation; Yuansheng Gao Book 20171st edition Springer Nature Singapore Pte Ltd.

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发表于 2025-3-21 19:41:05 | 显示全部楼层 |阅读模式
期刊全称Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation
影响因子2023Yuansheng Gao
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发行地址Providing a concise yet comprehensive review of the morphological, biochemical, electrical, mechanical, and metabolic properties of vascular smooth muscle.Focusing on the cutting-edge developments in
图书封面Titlebook: Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation;  Yuansheng Gao Book 20171st edition Springer Nature Singapore Pte Ltd.
影响因子.This book provides a concise yet comprehensive review of the morphological, biochemical, electrical, mechanical, and metabolic properties of vascular smooth muscle, the regulation of vascular activities and the intracellular signaling involved. It particularly focuses on recently identified vasoactive agents, enzymes and transduction mechanisms. It also discusses the latest findings in the regulation of cerebral, coronary and pulmonary circulation as well as vascular activity under hypoxia and ageing. The contraction and dilatation activities of vasculature are of fundamental importance for maintaining circulation homeostasis and adapting physiological changes. Over the last four decades, there have been significant advances in our understanding of the biochemical, structural, genetic, physiological, and pharmacological aspects of vascular activity regulation, and these insights into the responsiveness of blood vessels under normal and pathophysiological conditions help to provide valuable weapons in the fight vascular diseases. The book is of interest to researchers and graduate students, both in basic research and in clinic settings, in the field of vascular biology..
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Rechenbeispiele in der Transfusionsmedizin,ity in the tension development when its length is altered and in the shortening speed when its load is changed. This is ascribed to a dynamic process of formation of cross-bridge by myosins and actins and lends VSM marked flexibility to meet different physiological needs. The above topic will be dis
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Transfusionelle CMV-Problematik,oactive intestinal peptide, nitric oxide, calcitonin gene-related peptide, substance P, and others. Some of them are vasoconstrictors and some are vasodilators. The balance of the activities of these neurotransmitters is critical for the integrity of vascular function.
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https://doi.org/10.1007/978-3-211-89851-2sed Ca. influx. Among these vasodilators, NO is the dominant player, PGI. often has a complementary role, and EDHFs are more important in resistant arteries where NO is less crucial. The vasodilatory effects of these agents are counteracted by various vasoconstrictors in particular ET-1, which exert
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,Thrombozytopenischer Refraktärzustand,onse is initiated by the opening of mechanosensitive ion channels such as TRPC6 channel, TRPM4 channel, and probably a heteromultimeric channel complex formed by the β and γ subunits of epithelial sodium channels and the subunit 2 of acid-sensing ion channel. Myogenic response is of fundamental impo
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Book 20171st editione responsiveness of blood vessels under normal and pathophysiological conditions help to provide valuable weapons in the fight vascular diseases. The book is of interest to researchers and graduate students, both in basic research and in clinic settings, in the field of vascular biology..
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Architecture of the Blood Vesselswill be discussed. Furthermore, the gap junctions that form intercellular connections and the vessels that provide nutrient to vessel walls as well as some cell types of functional importance including fibroblasts, stem/progenitor cells, and perivascular adipose tissue will be reviewed.
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Vascular Endotheliumnhibitory actions on platelet aggregation and coagulation and its facilitating effect on fibrinolysis. These effects are mainly mediated by the endothelium-derived molecules such as nitric oxide, prostaglandin I., thrombomodulin, heparan sulfate proteoglycan, and tissue factor pathway inhibitor. How
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