Titlebook: Beta-Arrestins; Methods and Protocol Mark G. H. Scott,Stéphane A. Laporte Book 2019 Springer Science+Business Media, LLC, part of Springer

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Book 2019ng cellular and in vivo consequences that arise. The chapters in this book cover diverse topics around b-arrs such as their established roles in GPCR regulation and trafficking; regulatory scaffolding functions of b-arrs in  MAPK signaling, cAMP hydrolysis and cytoskeletal dynamics; proteomic analys

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https://doi.org/10.1057/9780230620131ranslocation of RalGDS is β-arrestin-dependent and can be inhibited by either the expression of the β-arrestin1 amino-terminal domain or the expression of RalGDS clone 284 (amino acid residues 616–768 of RalGDS). We describe here a methodology for assessing GPCR-dependent stimulation of RalGDS plasma membrane translocation.

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https://doi.org/10.1057/9780230620131amework. We explain how it can be applied to the understanding of β-arrestin-dependent signaling networks. We illustrate our methodology through the modeling of β-arrestin recruitment kinetics at the follicle-stimulating hormone (FSH) receptor supported by in-house bioluminescence resonance energy transfer (BRET) data.

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Workflow Description to Dynamically Model β-Arrestin Signaling Networksamework. We explain how it can be applied to the understanding of β-arrestin-dependent signaling networks. We illustrate our methodology through the modeling of β-arrestin recruitment kinetics at the follicle-stimulating hormone (FSH) receptor supported by in-house bioluminescence resonance energy transfer (BRET) data.

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