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Titlebook: Autoimmune Thyroiditis; Approaches Towards i W. A. Scherbaum,U. Bogner,G. F. Bottazzo Conference proceedings 1991 Springer-Verlag Berlin He

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Comparison Between Microsomal Antibodies by Immunofluorescence and Anti-TPO Antibodies in Healthy Ininical usefulness of replacing measurements of MAb by a commercial method for quantifying anti-TPO. Furthermore, we wanted to compare the anti-TPO measurements with an immunofluorescence method for MAb, which is generally considered rather specific.
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Further Clarification of the Significance of Thyroid Peroxidase Activity-Inhibiting Immunoglobulins n vitro, and that these TPO activity-inhibiting immunoglobulins (TPII) seem to inhibit thyroid function in some patients, but the relationship between TPII index and thyroid function is not simple. In this study, we examined whether TPII can inhibit the activity of TPO present in thyroid tissue.
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Comparison of Circulating Thyroid Microsomal, Cytotoxic and Thyroid Peroxidase Antibodiestibodies that are cytotoxic for thyroid cells in vitro have been found in the sera of patients with autoimmune thyroiditis and exhibiting antimicrosomal antibodies. These cytotoxic antibodies are active both directly in the presence of complement [2, 3, 4] and in association with K cells [5].
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Donald P. Goldstein,Ross S. Berkowitzon of thyroid hormones from thyroglobulin and comprises a 105-kDa protein [3] which has recently been cloned [4]. Two forms of TPO cDNA have been reported, termed TPO-1 and TPO-2, with exon 10 coding for 57 amino acids is spliced from the coding sequence of the shorter TPO-2 cDNA [5].
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https://doi.org/10.1007/978-3-642-49213-6mmunity, but also specific recognition by the T-cell receptor supplemented by helper factors is necessary for an immune response [2]. Since organ-specific autoimmune diseases are chronic diseases, it might he speculated that the helper factors (interleukins), which provide a positive feedback, play
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https://doi.org/10.1007/978-3-7091-8925-2n the blood group Kell and TPO autoantibody. Kell has been reported to be linked to the locus governing the ability to taste phenylthiocarbamide (PTC) [3] which in turn may be associated with certain forms of thyroid disease [4]. In order to explore further the genetic basis of autoimmune thyroid di
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Catherine Kim,Assiamira Ferraraods (passive hemagglutination and radioligand assay) with a newly developed radioimmunoassay for anti-TPO antibodies using the inhibition of binding of labeled TPO to a solid-phase bound monoclonal antibody by autoantibodies [3].
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