Titlebook: Autoantibodies in Neurological Diseases; Angela Vincent,Gianvito Martino Book 2002 Springer-Verlag Italia 2002 Antigen.Nervous System.Neur

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https://doi.org/10.1007/978-3-642-55921-1with neuroblastoma [.] and other neural-crest-derived tumours has been described in some children (paraneoplastic OMS, OMS-NB.), whilst several other potential causal relationships have also been found (Table 1), but little progress has been made in understanding the etiology and treatment of this s

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Wissensmanagement im Projektgeschäftune response. This weak response was explained by: (1) the presence of a blood-brain barrier (BBB); (2) constitutive lack or low expression of proteins of the immune system, such as major histocompatibility complex (MHC) class I and II, co-stimulatory and accessory molecules, on cells of the CNS (gU

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https://doi.org/10.1007/978-3-662-07470-1a, sustained inflammation sometimes compromises vessel integrity. The central nervous system (CNS) is the target organ of isolated, or primary, vasculitis (primary angiitis of the CNS, or PACNS). Far more frequently, the involvement of the CNS is part of a more complex scenario involving other organ

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https://doi.org/10.1007/978-88-470-2097-9Antigen; Nervous System; Neuromuscular disorders; antibodies; autoimmunity; inflammation; paraneoplastic s

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978-88-470-2163-1Springer-Verlag Italia 2002

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Introduction - Approaches to Understanding Immune-Mediated Neurological Disorders: Measuring and Ev junction disorders, immune-mediated demyelinating disorders, and also some form of epilepsy, stroke, Alzheimer’s and Parkinsons’ diseases, and prion diseases. It is, therefore, particularly important to distinguish those conditions which are caused by dysfunction of the immune system, from those in

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