Titlebook: Autoantibodies in Neurological Diseases; Angela Vincent,Gianvito Martino Book 2002 Springer-Verlag Italia 2002 Antigen.Nervous System.Neur

Diatribe

Ontogeny of Skeletal Muscle Cells,e early stages.Skeletal myogenesis begins shortly after gastrulation but persists, at least in mammals, until the end of postnatal growth, and the potential for myogenesis continues for the entire life span of the animal [.]. Local signalling commits mesodermal cells to a myogenic fate, and shortly

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hysterectomy

Diluge

Stiff-Man Syndrome: Pathogenetic, Nosological and Therapeutic Considerations, muscle spasms. It was first described by Moersch and Woltmann in 1956 [.]. A set of diagnostic criteria was proposed by Gordon et al. [.] and Lorish et al. [.]. Diagnostic criteria included: 1) a prodrome of stiffness and rigidity in axial muscles; 2) a slow progression of stiffness involving proxi

Alpha-Cells

Opsoclonus-Myoclonus Syndrome in Childhood,with neuroblastoma [.] and other neural-crest-derived tumours has been described in some children (paraneoplastic OMS, OMS-NB.), whilst several other potential causal relationships have also been found (Table 1), but little progress has been made in understanding the etiology and treatment of this s

paragon

Epilepsy and Autoantibodies,une response. This weak response was explained by: (1) the presence of a blood-brain barrier (BBB); (2) constitutive lack or low expression of proteins of the immune system, such as major histocompatibility complex (MHC) class I and II, co-stimulatory and accessory molecules, on cells of the CNS (gU

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Introduction - Approaches to Understanding Immune-Mediated Neurological Disorders: Measuring and Ev which immune responses play a purely secondary role. In this chapter, we first provide an abbreviated introduction to neuroimmunology, and then discuss ways of measuring immune responses to neuronal antigens, and of approaches that evaluate their pathogenic roles.

Adrenal-Glands

膝盖

Idiopathic Inflammatory Myopathies: Immunological Aspects,DM), polymyositis (PM) and inclusion body myositis (IBM). The latter includes sporadic (s-IBM) and hereditary inclusion body myopathy (h-IBM), which is an a hereditary progressive muscle disease with muscle pathology similar to the s-IBM,but lacking lymphocytic inflammation [.].