Titlebook: Antiarrhythmic Drugs; Mechanisms of Antiar Günter Breithardt,Martin Borggrefe,Gerhard Hindric Book 1995 Springer-Verlag Berlin Heidelberg 1

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pericardium

Die Physiologie der peripheren Nerventhe L type Ca channel (i.) and the T type Ca channel (i.), is vast. It is not the intent of this chapter to review this exhaustive literature; however, initially we describe some of the physiological and biophysical facts essential for the study of channel pharmacology. On the other hand, the litera

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Die Physiologie der „inneren Sekretion“rdium. Evidence has been advanced in support of the existence of at least four electrophysiologically distinct cell types in the canine ventricle including: epicardial, M, endocardial and Purkinje cells. A number of studies have described distinctions between ventricular epicardium and endocardium [

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Die Physiologie der peripheren Nervenrugs prolonging the action potential duration. The immediate reason was the Cardiac Arrhythmia Suppression Trial (CAST) study (1989), which showed an increased mortality in patients treated for premature ventricular extrasystoles following myocardial infarction with drugs representative of the class

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https://doi.org/10.1007/978-3-662-36786-5on is slow. With some delay it enters the His-Purkinje system which quickly activates the ventricular myocardium. When activation of the heart deviates from this normal activation sequence an arrhythmia results. The most serious arrhythmias are those where the normal pumping function of the heart is

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Peripheres vegetatives Nervensysteme accumulation of toxic metabolites, shifts in intracellular and extracellular ionic activities, and modulation of neurohumoral influences and autonomic reflexes, as well as direct damage to the cell membrane, its constituent channels, pumps and gap junctions, and the supporting collagen matrix. Pre