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Titlebook: Unusual Secretory Pathways: From Bacteria to Man; Karl Kuchler,Anna Rubartelli,Barry Holland Book 1997 Springer-Verlag Berlin Heidelberg 1

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书目名称Unusual Secretory Pathways: From Bacteria to Man
编辑Karl Kuchler,Anna Rubartelli,Barry Holland
视频video
丛书名称Molecular Biology Intelligence Unit
图书封面Titlebook: Unusual Secretory Pathways: From Bacteria to Man;  Karl Kuchler,Anna Rubartelli,Barry Holland Book 1997 Springer-Verlag Berlin Heidelberg 1
描述This book constitutes the first comprehensive discussion and collection of facts and functions of unconventional secretory pathways, some of which are of importance in human disease and pathology. The function of ABC transporters in these processes is also discussed. This comparison of common aspects and similarities in all living cells - from bacteria to man - is an excellent reference book with "textbook" character.
出版日期Book 1997
关键词bacteria; cells; pathology; protein; transport
版次1
doihttps://doi.org/10.1007/978-3-662-22581-3
isbn_softcover978-3-662-22583-7
isbn_ebook978-3-662-22581-3Series ISSN 1431-0414
issn_series 1431-0414
copyrightSpringer-Verlag Berlin Heidelberg 1997
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Protein Secretion in Gram-Negative Bacteria,lin system, are transported from the periplasm via a single outer membrane “translocator” protein; Type VI for the special case of the filamentous phage (nucleo-protein) secretory pathway. The secretion of flagellar proteins on to the cell surface, although not discussed in this review, could be con
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Mammalian ABC Transporters and Leaderless Secretion: Facts and Speculations,has not been possible so far to correlate the presence of sequence motifs in the transporter to a particular substrate or substrate class. Substrate assignment has proven to be the most difficult task in the analysis of mammalian ABC transporters, even in cases when functional clues are provided by
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The Transporters Associated with Antigen Processing (TAP),fection or malignant transformation a different set of peptides is displayed by MHC class I molecules. These antigen-loaded class I complexes are recognized by cytotoxic T cells via the T cell receptors as nonself, thus leading to the destruction of the abnormal cell.
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