Titlebook: Symposium on Neuroglia / Symposium Concernant La Neuroglie / Symposium über die Neuroglia; Papers Presented on F. Erbslöh,A. Oksche,F. Sei

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Gliazellen als Manifestationsorte von Stoffwechselkrankheiten,storage substances in experiments, especially in tissue cultures. The decision, what types of cells are damaged by the storage diseases, is determined by the physiological role played by the stored substances in the structure of the cell membranes.

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Pathologie der Makro- und Mikroglia im elektronenmikroskopischen Bild,crogliacells contain phagosomes of different types as an indication of the intensive uptake and decomposition of substances. A source of microgliacell proliferation might be the pericytes of the capillary walls.

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Zur Ultrastruktur der Astroglia und des Status spongiosus,ng an enlargement of the intercellular space system, and the . of the white matter (in cases of infantile diffuse spongioform degeneration) probably by an interlamellar hydrops of the myelin sheath, which is well known in a more excessive state in TET-poisoning.

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,Gliabefunde bei den diffusen und fokal-disseminierten Formen der spongiösen Neurodystrophien des fr Scherer was predominating, in the elder cases mature glia was abundant. Alzheimer-II-glia was missing..In the focal disseminating cases one could find only occasionally myelinisation glia. The reaction of the mature glia did not differ from glia reaction of the adult brain.

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Die Hirncapillaren und ihre Glia,t of the vessels in various regions of various species. The general principle is the “hermetism” of the network-capillaries and of the loopings. Both network and looping have no pores, they have a closed basement membrane and a complete glia sheet. In chambers of the basement membrane can be found f

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